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American Journal of Respiratory Cell and Molecular Biology|August 30, 2014
Senescence-associated secretory phenotype and its possible role in chronic obstructive pulmonary diseaseManish Kumar, Werner Seeger, Robert VoswinckelExperimental Hematology|July 24, 2002
The protein tyrosine kinase inhibitor SU5614 inhibits VEGF-induced endothelial cell sprouting and induces growth arrest and apoptosis by inhibition of c-kit in AML cellsKarsten Spiekermann, Florian Faber, Robert Voswinckel, et al.Drug Design, Development and Therapy|February 1, 2012
Inhaled treprostinil: a therapeutic reviewRichard N Channick, Robert Voswinckel, Lewis J RubinHerz|June 21, 2005
[Emerging therapies for the treatment of pulmonary arterial hypertension]Hossein Ardeschir Ghofrani, Robert Voswinckel, Frank Reichenberger, et al.Journal of Anatomy|November 15, 2011
Myocardial remodelling in left ventricular atrophy induced by caloric restrictionCarina Gruber, Nadine Nink, Sandeep Nikam, et al.Circulation Research|December 6, 2003
Bone marrow-derived cells do not incorporate into the adult growing vasculatureTibor Ziegelhoeffer, Borja Fernandez, Sawa Kostin, et al.Journal of the American College of Cardiology|October 3, 2006
Safety and efficacy of inhaled treprostinil as add-on therapy to bosentan in pulmonary arterial hypertensionRichard N Channick, Horst Olschewski, Werner Seeger, et al.American Journal of Respiratory Cell and Molecular Biology|February 1, 2011
Treprostinil inhibits the adhesion and differentiation of fibrocytes via the cyclic adenosine monophosphate-dependent and Ras-proximate protein-dependent inactivation of extracellular regulated kinaseVandana S Nikam, Gesa Wecker, Ralph Schermuly, et al.Anesthesia and Analgesia|September 16, 2010
Perioperative mortality in patients with pulmonary hypertension undergoing major joint replacementStavros G Memtsoudis, Yan Ma, Ya Lin Chiu, et al.Future Cardiology|March 17, 2010
Riociguat for pulmonary hypertensionHossein-Ardeschir Ghofrani, Robert Voswinckel, Henning Gall, et al.Pageof 7