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Trends in Endocrinology and Metabolism: TEM|April 9, 2015
Mitochondria in autoinflammation: cause, mediator or bystander?Robert van der Burgh, Marianne BoesClinical Immunology (Orlando, Fla.)|November 1, 2012
Mevalonate kinase deficiency, a metabolic autoinflammatory diseaseRobert van der Burgh, Nienke M Ter Haar, Marianne L Boes, et al.The Journal of Biological Chemistry|August 10, 2014
Unprenylated RhoA contributes to IL-1β hypersecretion in mevalonate kinase deficiency model through stimulation of Rac1 activityRobert van der Burgh, Kalliopi Pervolaraki, Marjolein Turkenburg, et al.Oncotarget|March 19, 2016
Reduced serpinB9-mediated caspase-1 inhibition can contribute to autoinflammatory diseaseRobert van der Burgh, Jan Meeldijk, Lieneke Jongeneel, et al.The Journal of Biological Chemistry|December 21, 2013
Defects in mitochondrial clearance predispose human monocytes to interleukin-1β hypersecretionRobert van der Burgh, Lotte Nijhuis, Kalliopi Pervolaraki, et al.Pageof 1