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Trends in Endocrinology and Metabolism: TEM|April 9, 2015
Mitochondria in autoinflammation: cause, mediator or bystander?Robert van der Burgh, Marianne BoesThe Journal of Biological Chemistry|August 10, 2014
Unprenylated RhoA contributes to IL-1β hypersecretion in mevalonate kinase deficiency model through stimulation of Rac1 activityRobert van der Burgh, Kalliopi Pervolaraki, Marjolein Turkenburg, et al.Oncotarget|March 19, 2016
Reduced serpinB9-mediated caspase-1 inhibition can contribute to autoinflammatory diseaseRobert van der Burgh, Jan Meeldijk, Lieneke Jongeneel, et al.The Journal of Biological Chemistry|December 21, 2013
Defects in mitochondrial clearance predispose human monocytes to interleukin-1β hypersecretionRobert van der Burgh, Lotte Nijhuis, Kalliopi Pervolaraki, et al.Clinical Immunology (Orlando, Fla.)|November 1, 2012
Mevalonate kinase deficiency, a metabolic autoinflammatory diseaseRobert van der Burgh, Nienke M Ter Haar, Marianne L Boes, et al.Nature|July 9, 2004
Translating cell biology in vitro to immunity in vivoMarianne Boes, Hidde L PloeghCancer Letters|February 21, 2015
TLR3 triggering regulates PD-L1 (CD274) expression in human neuroblastoma cellsMarianne Boes, Friederike Meyer-WentrupCommunicative & Integrative Biology|October 20, 2015
MICAL-L1-related and unrelated mechanisms underlying elongated tubular endosomal network (ETEN) in human dendritic cellsEwoud B Compeer, Marianne BoesTrends in Cancer|October 25, 2018
NFκB and MHC-1 Interplay in Neuroblastoma and ImmunotherapyLotte Spel, Ariën Schiepers, Marianne BoesJournal of Immunology (Baltimore, Md. : 1950)|March 9, 2004
Myeloid differentiation factor 88 is required for cross-priming in vivoDeborah Palliser, Hidde Ploegh, Marianne BoesPageof 12