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Nature|June 26, 2007
Ambra1 regulates autophagy and development of the nervous systemGian Maria Fimia, Anastassia Stoykova, Alessandra Romagnoli, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 17, 2025
Deletion of p63 exon 13 in mice reveals C-terminal isoform-specific functions in epithelial developmentAnna Maria Lena, Artem Smirnov, Mara Mancini, et al.
Cells|June 2, 2021
High Levels of TRIM5α Are Associated with Xenophagy in HIV-1-Infected Long-Term NonprogressorsFabiola Ciccosanti, Marco Corazzari, Rita Casetti, et al.
Cardiovascular Pathology : the Official Journal of the Society for Cardiovascular Pathology|December 1, 2020
Fatal Takotsubo syndrome in critical COVID-19 related pneumoniaLuca Titi, Eugenia Magnanimi, Massimo Mancone, et al.
Frontiers in Molecular Biosciences|December 13, 2021
Pleural Mesothelial Cells Modulate the Inflammatory/Profibrotic Response During SARS-CoV-2 InfectionGiulia Matusali, Flavia Trionfetti, Veronica Bordoni, et al.
The Journal of Dermatology|February 24, 2021
Dermatological manifestations during COVID-19 and histological picture: Description of two clinical casesPaola Mencarini, Alessandra Scarabello, Franca Del Nonno, et al.
The American Journal of Pathology|March 26, 2003
Transglutaminase type II plays a protective role in hepatic injuryRoberta Nardacci, Oreste Lo Iacono, Fabiola Ciccosanti, et al.
The Journal of Cell Biology|October 6, 2010
The dynamic interaction of AMBRA1 with the dynein motor complex regulates mammalian autophagySabrina Di Bartolomeo, Marco Corazzari, Francesca Nazio, et al.
The Journal of Infectious Diseases|September 11, 2020
Postmortem Findings in Italian Patients With COVID-19: A Descriptive Full Autopsy Study of Cases With and Without ComorbiditiesLaura Falasca, Roberta Nardacci, Daniele Colombo, et al.
Cardiovascular Research|March 11, 2021
Human cardiosphere-derived stromal cells exposed to SARS-CoV-2 evolve into hyper-inflammatory/pro-fibrotic phenotype and produce infective viral particles depending on the levels of ACE2 receptor expressionAlessandra Amendola, Gloria Garoffolo, Paola Songia, et al.
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