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The Journal of Biological Chemistry|June 13, 2003
Antagonistic effects of beta-site amyloid precursor protein-cleaving enzymes 1 and 2 on beta-amyloid peptide production in cellsGuriqbal Basi, Normand Frigon, Robin Barbour, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 3, 2006
Increased T cell recruitment to the CNS after amyloid beta 1-42 immunization in Alzheimer's mice overproducing transforming growth factor-beta 1Marion S Buckwalter, Bronwen S Coleman, Manuel Buttini, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 9, 2007
Active beta-amyloid immunization restores spatial learning in PDAPP mice displaying very low levels of beta-amyloidGuiquan Chen, Karen S Chen, Dione Kobayashi, et al.
The American Journal of Pathology|August 28, 2004
Morphological characterization of Thioflavin-S-positive amyloid plaques in transgenic Alzheimer mice and effect of passive Abeta immunotherapy on their clearanceThierry Bussière, Frédérique Bard, Robin Barbour, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 4, 2008
Immunotherapy reduces vascular amyloid-beta in PDAPP miceSally Schroeter, Karen Khan, Robin Barbour, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 7, 2005
Beta-amyloid immunotherapy prevents synaptic degeneration in a mouse model of Alzheimer's diseaseManuel Buttini, Eliezer Masliah, Robin Barbour, et al.
Biochemical and Biophysical Research Communications|September 28, 2013
Thermodynamic and kinetic characterization of hydroxyethylamine β-secretase-1 inhibitorsKalyani Mondal, Karin Regnstrom, Winse Morishige, et al.
Plos One|January 10, 2013
AL amyloid imaging and therapy with a monoclonal antibody to a cryptic epitope on amyloid fibrilsJonathan S Wall, Stephen J Kennel, Angela Williams, et al.
The American Journal of Pathology|January 15, 2013
Axonopathy in an α-synuclein transgenic model of Lewy body disease is associated with extensive accumulation of C-terminal-truncated α-synucleinDora Games, Peter Seubert, Edward Rockenstein, et al.
Neuro-Degenerative Diseases|January 10, 2008
Red blood cells are the major source of alpha-synuclein in bloodRobin Barbour, Kristin Kling, John P Anderson, et al.
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