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Robyn Cunard

Showing results (1-10 of 30) with videos related to

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Current Hypertension Reports|July 19, 2017
Endoplasmic Reticulum Stress, a Driver or an Innocent Bystander in Endothelial Dysfunction Associated with Hypertension?Robyn Cunard
Scientifica|February 4, 2014
Mammalian tribbles homologs at the crossroads of endoplasmic reticulum stress and Mammalian target of rapamycin pathwaysRobyn Cunard
Current Opinion in Investigational Drugs (London, England : 2000)|May 26, 2005
The potential use of PPARalpha agonists as immunosuppressive agentsRobyn Cunard
Journal of Clinical Medicine|August 5, 2015
Endoplasmic Reticulum Stress in the Diabetic Kidney, the Good, the Bad and the UglyRobyn Cunard
American Journal of Physiology. Renal Physiology|February 25, 2011
The endoplasmic reticulum stress response and diabetic kidney diseaseRobyn Cunard, Kumar Sharma
The Journal of Allergy and Clinical Immunology|February 20, 2003
18. Immune-mediated renal diseaseRobyn Cunard, Carolyn J Kelly
Journal of the American Society of Nephrology : JASN|April 19, 2002
T cells and minimal change diseaseRobyn Cunard, Carolyn J Kelly
Contributions to Nephrology|June 11, 2011
Antifibrotic treatment and other new strategies for improving renal outcomesAnna Mathew, Robyn Cunard, Kumar Sharma
Molecular Immunology|September 15, 2009
PPARalpha ligands cause lymphocyte depletion and cell cycle block and this is associated with augmented TRB3 and reduced Cyclin B1 expressionElizabeth Morse, Erin Selim, Robyn Cunard
Molecular Immunology|September 5, 2006
Fibrates upregulate TRB3 in lymphocytes independent of PPAR alpha by augmenting CCAAT/enhancer-binding protein beta (C/EBP beta) expressionErin Selim, Julie T Frkanec, Robyn Cunard
Pageof 3

Showing results (1-10 of 30) with videos related to

Sort By:
Pageof 3
Current Hypertension Reports|July 19, 2017
Endoplasmic Reticulum Stress, a Driver or an Innocent Bystander in Endothelial Dysfunction Associated with Hypertension?Robyn Cunard
Scientifica|February 4, 2014
Mammalian tribbles homologs at the crossroads of endoplasmic reticulum stress and Mammalian target of rapamycin pathwaysRobyn Cunard
Current Opinion in Investigational Drugs (London, England : 2000)|May 26, 2005
The potential use of PPARalpha agonists as immunosuppressive agentsRobyn Cunard
Journal of Clinical Medicine|August 5, 2015
Endoplasmic Reticulum Stress in the Diabetic Kidney, the Good, the Bad and the UglyRobyn Cunard
American Journal of Physiology. Renal Physiology|February 25, 2011
The endoplasmic reticulum stress response and diabetic kidney diseaseRobyn Cunard, Kumar Sharma
The Journal of Allergy and Clinical Immunology|February 20, 2003
18. Immune-mediated renal diseaseRobyn Cunard, Carolyn J Kelly
Journal of the American Society of Nephrology : JASN|April 19, 2002
T cells and minimal change diseaseRobyn Cunard, Carolyn J Kelly
Contributions to Nephrology|June 11, 2011
Antifibrotic treatment and other new strategies for improving renal outcomesAnna Mathew, Robyn Cunard, Kumar Sharma
Molecular Immunology|September 15, 2009
PPARalpha ligands cause lymphocyte depletion and cell cycle block and this is associated with augmented TRB3 and reduced Cyclin B1 expressionElizabeth Morse, Erin Selim, Robyn Cunard
Molecular Immunology|September 5, 2006
Fibrates upregulate TRB3 in lymphocytes independent of PPAR alpha by augmenting CCAAT/enhancer-binding protein beta (C/EBP beta) expressionErin Selim, Julie T Frkanec, Robyn Cunard
Pageof 3