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Cancers|November 12, 2013
Do non-genomically encoded fusion transcripts cause recurrent chromosomal translocations?Eric Kowarz, Theo Dingermann, Rolf MarschalekOncogene|June 6, 2003
MLL-mediated transcriptional gene regulation investigated by gene expression profilingDorothe Schraets, Tanja Lehmann, Theo Dingermann, et al.American Journal of Blood Research|March 21, 2012
Premature transcript termination, trans-splicing and DNA repair: a vicious path to cancerEric Kowarz, Jennifer Merkens, Michael Karas, et al.Nucleic Acids Research|April 30, 2010
Transcriptional properties of human NANOG1 and NANOG2 in acute leukemic cellsIrina Eberle, Birgit Pless, Miriam Braun, et al.Oncogene|June 29, 2004
Interaction of AF4 wild-type and AF4.MLL fusion protein with SIAH proteins: indication for t(4;11) pathobiology?Adelheid Bursen, Sven Moritz, Anne Gaussmann, et al.European Journal of Haematology|June 30, 2009
Two independent gene signatures in pediatric t(4;11) acute lymphoblastic leukemia patientsLuca Trentin, Marco Giordan, Theo Dingermann, et al.Biotechnology Journal|August 8, 2006
Genomic DNA of leukemic patients: target for clinical diagnosis of MLL rearrangementsClaus Meyer, Eric Kowarz, Björn Schneider, et al.Blood|March 3, 2010
The AF4.MLL fusion protein is capable of inducing ALL in mice without requirement of MLL.AF4Adelheid Bursen, Karen Schwabe, Brigitte Rüster, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|February 4, 2009
An interstitial deletion at 3p21.3 results in the genetic fusion of MLH1 and ITGA9 in a Lynch syndrome familyClaus Meyer, Angela Brieger, Guido Plotz, et al.Biotechnology Journal|November 28, 2007
Recombinant therapeutic proteins: production platforms and challengesTheo DingermannPageof 16