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Atherosclerosis. Supplements|December 11, 2003
Hyperhomocysteinemia, endothelial dysfunction, and cardiovascular risk: the potential role of ADMASteven R Lentz, Roman N Rodionov, Sanjana DayalTrends in Pharmacological Sciences|October 9, 2014
AGXT2: a promiscuous aminotransferaseRoman N Rodionov, Natalia Jarzebska, Norbert Weiss, et al.The Journal of Biological Chemistry|December 19, 2009
Human alanine-glyoxylate aminotransferase 2 lowers asymmetric dimethylarginine and protects from inhibition of nitric oxide productionRoman N Rodionov, Daryl J Murry, Sarah F Vaulman, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|March 15, 2019
Is Homoarginine a Protective Cardiovascular Risk Factor?Ekaterina S Karetnikova, Natalia Jarzebska, Alexander G Markov, et al.Stroke|December 8, 2007
Overexpression of dimethylarginine dimethylaminohydrolase inhibits asymmetric dimethylarginine-induced endothelial dysfunction in the cerebral circulationHayan Dayoub, Roman N Rodionov, Cynthia Lynch, et al.American Journal of Physiology. Heart and Circulatory Physiology|June 24, 2008
Tissue-specific downregulation of dimethylarginine dimethylaminohydrolase in hyperhomocysteinemiaSanjana Dayal, Roman N Rodionov, Erland Arning, et al.Hematology. American Society of Hematology. Education Program|December 4, 2016
Thrombosis in the setting of obesity or inflammatory bowel diseaseSteven R LentzBlood|November 19, 2016
Thrombosis in the setting of obesity or inflammatory bowel diseaseSteven R LentzVASA. Zeitschrift Fur Gefasskrankheiten|October 24, 2014
Medical management of abdominal aortic aneurysmsNorbert Weiss, Roman N Rodionov, Adrian MahlmannVascular Medicine (London, England)|February 1, 2006
ADMA and hyperhomocysteinemiaSanjana Dayal, Steven R LentzPageof 18