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Rona K Graham

Showing results (11-20 of 44) with videos related to

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The European Journal of Neuroscience|September 22, 2021
Diet enriched in omega-3 fatty acids alleviates olfactory system deficits in APOE4 transgenic miceMelissa Lessard-Beaudoin, Laura M Gonzalez, Majed AlOtaibi, et al.
Brain : a Journal of Neurology|February 20, 2009
Prevention of depressive behaviour in the YAC128 mouse model of Huntington disease by mutation at residue 586 of huntingtinMahmoud A Pouladi, Rona K Graham, Joanna M Karasinska, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 29, 2010
Phosphorylation of huntingtin at Ser421 in YAC128 neurons is associated with protection of YAC128 neurons from NMDA-mediated excitotoxicity and is modulated by PP1 and PP2AMartina Metzler, Lu Gan, Gelareh Mazarei, et al.
Circulation. Arrhythmia and Electrophysiology|June 13, 2015
Prolongation of action potential duration and QT interval during epilepsy linked to increased contribution of neuronal sodium channels to cardiac late Na+ current: potential mechanism for sudden death in epilepsyMichael Biet, Nathalie Morin, Melissa Lessard-Beaudoin, et al.
Neurobiology of Disease|September 17, 2005
Striatal neuronal apoptosis is preferentially enhanced by NMDA receptor activation in YAC transgenic mouse model of Huntington diseaseJacqueline Shehadeh, Herman B Fernandes, Melinda M Zeron Mullins, et al.
Journal of Huntington'S Disease|July 27, 2014
Caspase-6-Resistant Mutant Huntingtin Does not Rescue the Toxic Effects of Caspase-Cleavable Mutant Huntingtin in vivoRona K Graham, Yu Deng, Mahmoud A Pouladi, et al.
Neurobiology of Disease|February 6, 2007
Phenotypic abnormalities in the YAC128 mouse model of Huntington disease are penetrant on multiple genetic backgrounds and modulated by strainJeremy M Van Raamsdonk, Martina Metzler, Elizabeth Slow, et al.
Human Molecular Genetics|May 1, 2008
Activated caspase-6 and caspase-6-cleaved fragments of huntingtin specifically colocalize in the nucleusSimon C Warby, Crystal N Doty, Rona K Graham, et al.
Biogerontology|July 29, 2016
Age-dependent differential expression of death-associated protein 6 (Daxx) in various peripheral tissues and different brain regions of C57BL/6 male miceMélissa Lessard-Beaudoin, Mélissa Laroche, Marie-Josée Demers, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 28, 2008
Polyglutamine-modulated striatal calpain activity in YAC transgenic huntington disease mouse model: impact on NMDA receptor function and toxicityCatherine M Cowan, Mannie M Y Fan, Jing Fan, et al.
Pageof 5

Showing results (11-20 of 44) with videos related to

Sort By:
Pageof 5
The European Journal of Neuroscience|September 22, 2021
Diet enriched in omega-3 fatty acids alleviates olfactory system deficits in APOE4 transgenic miceMelissa Lessard-Beaudoin, Laura M Gonzalez, Majed AlOtaibi, et al.
Brain : a Journal of Neurology|February 20, 2009
Prevention of depressive behaviour in the YAC128 mouse model of Huntington disease by mutation at residue 586 of huntingtinMahmoud A Pouladi, Rona K Graham, Joanna M Karasinska, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 29, 2010
Phosphorylation of huntingtin at Ser421 in YAC128 neurons is associated with protection of YAC128 neurons from NMDA-mediated excitotoxicity and is modulated by PP1 and PP2AMartina Metzler, Lu Gan, Gelareh Mazarei, et al.
Circulation. Arrhythmia and Electrophysiology|June 13, 2015
Prolongation of action potential duration and QT interval during epilepsy linked to increased contribution of neuronal sodium channels to cardiac late Na+ current: potential mechanism for sudden death in epilepsyMichael Biet, Nathalie Morin, Melissa Lessard-Beaudoin, et al.
Neurobiology of Disease|September 17, 2005
Striatal neuronal apoptosis is preferentially enhanced by NMDA receptor activation in YAC transgenic mouse model of Huntington diseaseJacqueline Shehadeh, Herman B Fernandes, Melinda M Zeron Mullins, et al.
Journal of Huntington'S Disease|July 27, 2014
Caspase-6-Resistant Mutant Huntingtin Does not Rescue the Toxic Effects of Caspase-Cleavable Mutant Huntingtin in vivoRona K Graham, Yu Deng, Mahmoud A Pouladi, et al.
Neurobiology of Disease|February 6, 2007
Phenotypic abnormalities in the YAC128 mouse model of Huntington disease are penetrant on multiple genetic backgrounds and modulated by strainJeremy M Van Raamsdonk, Martina Metzler, Elizabeth Slow, et al.
Human Molecular Genetics|May 1, 2008
Activated caspase-6 and caspase-6-cleaved fragments of huntingtin specifically colocalize in the nucleusSimon C Warby, Crystal N Doty, Rona K Graham, et al.
Biogerontology|July 29, 2016
Age-dependent differential expression of death-associated protein 6 (Daxx) in various peripheral tissues and different brain regions of C57BL/6 male miceMélissa Lessard-Beaudoin, Mélissa Laroche, Marie-Josée Demers, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 28, 2008
Polyglutamine-modulated striatal calpain activity in YAC transgenic huntington disease mouse model: impact on NMDA receptor function and toxicityCatherine M Cowan, Mannie M Y Fan, Jing Fan, et al.
Pageof 5