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Science Translational Medicine|October 18, 2013
Inhibition of the cardiomyocyte-specific kinase TNNI3K limits oxidative stress, injury, and adverse remodeling in the ischemic heartRonald J Vagnozzi, Gregory J Gatto, Lara S Kallander, et al.
Circulation Research|April 11, 2014
Sorafenib cardiotoxicity increases mortality after myocardial infarctionJason M Duran, Catherine A Makarewich, Danielle Trappanese, et al.
Endocrinology|December 13, 2025
Activity of Cardiomyocyte Type 3 Deiodinase After Myocardial Infarction Influences Cardiac Recovery in FemalesMaigen Bethea, Tyler Cook, Preston Stafford, et al.
Circulation Research|December 4, 2024
Transcription Factor 21 Regulates Cardiac Myofibroblast Formation and FibrosisAnne Katrine Z Johansen, Rajesh K Kasam, Ronald J Vagnozzi, et al.
Nature Communications|September 12, 2020
Ontogeny of arterial macrophages defines their functions in homeostasis and inflammationTobias Weinberger, Dena Esfandyari, Denise Messerer, et al.
Circulation Research|July 28, 2016
Acute Catecholamine Exposure Causes Reversible Myocyte Injury Without Cardiac RegenerationMarkus Wallner, Jason M Duran, Sadia Mohsin, et al.
Biorxiv : the Preprint Server for Biology|January 30, 2023
Chromatin Remodeling Drives Immune-Fibroblast Crosstalk in Heart Failure PathogenesisMichael Alexanian, Arun Padmanabhan, Tomohiro Nishino, et al.
Nature|October 24, 2024
Chromatin remodelling drives immune cell-fibroblast communication in heart failureMichael Alexanian, Arun Padmanabhan, Tomohiro Nishino, et al.
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