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The Journal of Biological Chemistry|May 11, 2007
Greater bone formation of Y2 knockout mice is associated with increased osteoprogenitor numbers and altered Y1 receptor expressionPernilla Lundberg, Susan J Allison, Nicola J Lee, et al.Neuropeptides|April 1, 2014
Double deletion of orexigenic neuropeptide Y and dynorphin results in paradoxical obesity in miceAmy D Nguyen, Katy Slack, Christoph Schwarzer, et al.Plos One|January 30, 2016
Prader-Willi Critical Region, a Non-Translated, Imprinted Central Regulator of Bone Mass: Possible Role in Skeletal Abnormalities in Prader-Willi SyndromeEe-Cheng Khor, Bruce Fanshawe, Yue Qi, et al.Obesity (Silver Spring, Md.)|May 7, 2011
Peripheral-specific y2 receptor knockdown protects mice from high-fat diet-induced obesityYan-Chuan Shi, Shu Lin, Lesley Castillo, et al.Cell Metabolism|February 12, 2013
Arcuate NPY controls sympathetic output and BAT function via a relay of tyrosine hydroxylase neurons in the PVNYan-Chuan Shi, Jackie Lau, Zhou Lin, et al.Neuropeptides|October 16, 2012
The endogenous opioid dynorphin is required for normal bone homeostasis in micePaul A Baldock, Frank Driessler, Shu Lin, et al.Plos One|July 9, 2010
NPY neuron-specific Y2 receptors regulate adipose tissue and trabecular bone but not cortical bone homeostasis in miceYan-Chuan Shi, Shu Lin, Iris P L Wong, et al.The Journal of Biological Chemistry|May 11, 2007
Novel role of Y1 receptors in the coordinated regulation of bone and energy homeostasisPaul A Baldock, Susan J Allison, Pernilla Lundberg, et al.Nature Medicine|November 6, 2007
Tumor-induced anorexia and weight loss are mediated by the TGF-beta superfamily cytokine MIC-1Heiko Johnen, Shu Lin, Tamara Kuffner, et al.The Journal of Biological Chemistry|February 15, 2018
High dietary fat and sucrose results in an extensive and time-dependent deterioration in health of multiple physiological systems in miceJames G Burchfield, Melkam A Kebede, Christopher C Meoli, et al.Pageof 5