Showing results (1-10 of 16) with videos related to
Sort By:
Pageof 2
Plos One|September 19, 2015
TBX1 Represses Vegfr2 Gene Expression and Enhances the Cardiac Fate of VEGFR2+ CellsGabriella Lania, Rosa Ferrentino, Antonio BaldiniBMC Cell Biology|April 9, 2004
Mig12, a novel Opitz syndrome gene product partner, is expressed in the embryonic ventral midline and co-operates with Mid1 to bundle and stabilize microtubulesCaterina Berti, Bianca Fontanella, Rosa Ferrentino, et al.Human Mutation|January 16, 2007
MID1 mutation screening in a large cohort of Opitz G/BBB syndrome patients: twenty-nine novel mutations identifiedRosa Ferrentino, Maria Teresa Bassi, David Chitayat, et al.Disease Models & Mechanisms|February 20, 2021
EZH2 is required for parathyroid and thymic development through differentiation of the third pharyngeal pouch endodermCinzia Caprio, Gabriella Lania, Marchesa Bilio, et al.Human Molecular Genetics|February 28, 2012
Tbx1 is a negative modulator of Mef2cLuna Simona Pane, Zhen Zhang, Rosa Ferrentino, et al.Life Science Alliance|May 29, 2025
Tbx1 plays a critical role in focal adhesion dynamics through paxillin regulationOlimpia Iacolare, Rosa Ferrentino, Alessandra Altomonte, et al.Plos Genetics|March 23, 2012
Transcriptional control in cardiac progenitors: Tbx1 interacts with the BAF chromatin remodeling complex and regulates Wnt5aLi Chen, Filomena Gabriella Fulcoli, Rosa Ferrentino, et al.Human Molecular Genetics|August 16, 2013
Tbx1 regulates brain vascularizationSara Cioffi, Stefania Martucciello, Filomena Gabriella Fulcoli, et al.Biochemical and Biophysical Research Communications|May 15, 2024
Significant improvement of cardiac outflow tract septation defects in a DiGeorge syndrome model after minoxidil treatmentIlaria Aurigemma, Rosa Ferrentino, Varsha Poondi Krishnan, et al.Disease Models & Mechanisms|September 1, 2018
Tbx1 represses Mef2c gene expression and is correlated with histone 3 deacetylation of the anterior heart field enhancerLuna Simona Pane, Filomena Gabriella Fulcoli, Andrea Cirino, et al.Pageof 2