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The Lancet. Neurology|December 17, 2008
Genetic risk factors for variant Creutzfeldt-Jakob disease: a genome-wide association studySimon Mead, Mark Poulter, James Uphill, et al.Movement Disorders : Official Journal of the Movement Disorder Society|April 16, 2016
Nomenclature of genetic movement disorders: Recommendations of the international Parkinson and movement disorder society task forceConnie Marras, Anthony Lang, Bart P van de Warrenburg, et al.Neuroimage. Clinical|November 2, 2013
Evaluation of multi-modal, multi-site neuroimaging measures in Huntington's disease: Baseline results from the PADDINGTON studyNicola Z Hobbs, James H Cole, Ruth E Farmer, et al.Neurology|December 24, 2013
C9orf72 expansions are the most common genetic cause of Huntington disease phenocopiesDavina J Hensman Moss, Mark Poulter, Jon Beck, et al.Journal of the International Neuropsychological Society : JINS|May 24, 2016
Visuospatial Processing Deficits Linked to Posterior Brain Regions in Premanifest and Early Stage Huntington's DiseaseIzelle Labuschagne, Amy Mulick Cassidy, Rachael I Scahill, et al.The Journal of Clinical Investigation|September 22, 2012
Mutant huntingtin fragmentation in immune cells tracks Huntington's disease progressionAndreas Weiss, Ulrike Träger, Edward J Wild, et al.Molecular Cell|May 1, 2007
Disease-associated prion protein oligomers inhibit the 26S proteasomeMark Kristiansen, Pelagia Deriziotis, Derek E Dimcheff, et al.Neuropsychologia|November 1, 2015
The impact of occipital lobe cortical thickness on cognitive task performance: An investigation in Huntington's DiseaseEileanoir B Johnson, Elin M Rees, Izelle Labuschagne, et al.Journal of Huntington'S Disease|July 27, 2014
The potential of composite cognitive scores for tracking progression in Huntington's diseaseRebecca Jones, Julie C Stout, Izelle Labuschagne, et al.Journal of Huntington'S Disease|October 8, 2015
Neuropsychiatry and White Matter Microstructure in Huntington's DiseaseSarah Gregory, Rachael I Scahill, Kiran K Seunarine, et al.Pageof 32