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Nature Communications|April 9, 2025
The regulatory architecture of the primed pluripotent cell stateBo I Li, Mariano J Alvarez, Hui Zhao, et al.Cancer Research|September 18, 2020
CRISPR/Cas9-Mediated Point Mutation in Nkx3.1 Prolongs Protein Half-Life and Reverses Effects Nkx3.1 Allelic LossCai Bowen, Maho Shibata, Hailan Zhang, et al.Development (Cambridge, England)|November 18, 2005
Non-cell-autonomous role for Cripto in axial midline formation during vertebrate embryogenesisJianhua Chu, Jixiang Ding, Katherine Jeays-Ward, et al.Cell Stem Cell|March 10, 2015
Transient pairing of homologous Oct4 alleles accompanies the onset of embryonic stem cell differentiationMegan S Hogan, David-Emlyn Parfitt, Cinthya J Zepeda-Mendoza, et al.Developmental Dynamics : an Official Publication of the American Association of Anatomists|September 26, 2008
Mouse Fem1b interacts with the Nkx3.1 homeoprotein and is required for proper male secondary sexual developmentXi Wang, Nishita Desai, Ya-Ping Hu, et al.Genes & Development|February 18, 2016
Atg7 cooperates with Pten loss to drive prostate cancer tumor growthUrmila Santanam, Whitney Banach-Petrosky, Cory Abate-Shen, et al.Genome Medicine|January 17, 2025
Meta-analyses of mouse and human prostate single-cell transcriptomes reveal widespread epithelial plasticity in tissue regression, regeneration, and cancerLuis Aparicio, Laura Crowley, John R Christin, et al.The Prostate|October 13, 2012
Mash1 expression is induced in neuroendocrine prostate cancer upon the loss of Foxa2Aparna Gupta, Xiuping Yu, Tom Case, et al.Development (Cambridge, England)|October 5, 2007
Sulfated glycosaminoglycans are necessary for Nodal signal transmission from the node to the left lateral plate in the mouse embryoShinya Oki, Ryuju Hashimoto, Yuko Okui, et al.Proceedings of the National Academy of Sciences of the United States of America|September 16, 2006
Combinatorial activities of Akt and B-Raf/Erk signaling in a mouse model of androgen-independent prostate cancerHui Gao, Xuesong Ouyang, Whitney A Banach-Petrosky, et al.Pageof 14