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Science (New York, N.Y.)|August 9, 2014
C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteinsSarah Mizielinska, Sebastian Grönke, Teresa Niccoli, et al.
Brain : a Journal of Neurology|September 13, 2011
Clinical and neuroanatomical signatures of tissue pathology in frontotemporal lobar degenerationJonathan D Rohrer, Tammaryn Lashley, Jonathan M Schott, et al.
Nature Neuroscience|February 25, 2025
Neuronal polyunsaturated fatty acids are protective in ALS/FTDAshling Giblin, Alexander J Cammack, Niek Blomberg, et al.
Nucleic Acids Research|June 2, 2020
FUS ALS-causative mutations impair FUS autoregulation and splicing factor networks through intron retentionJack Humphrey, Nicol Birsa, Carmelo Milioto, et al.
Brain : a Journal of Neurology|July 15, 2011
A comparative clinical, pathological, biochemical and genetic study of fused in sarcoma proteinopathiesTammaryn Lashley, Jonathan D Rohrer, Rina Bandopadhyay, et al.
Acta Neuropathologica Communications|September 17, 2024
Molecular pathology, developmental changes and synaptic dysfunction in (pre-) symptomatic human C9ORF72-ALS/FTD cerebral organoidsAstrid T van der Geest, Channa E Jakobs, Tijana Ljubikj, et al.
Neurobiology of Aging|September 3, 2014
Screening a UK amyotrophic lateral sclerosis cohort provides evidence of multiple origins of the C9orf72 expansionPietro Fratta, James M Polke, Jia Newcombe, et al.
Biorxiv : the Preprint Server for Biology|July 15, 2024
The Neurolipid Atlas: a lipidomics resource for neurodegenerative diseases uncovers cholesterol as a regulator of astrocyte reactivity impaired by ApoE4Femke M Feringa, Sascha J Koppes-den Hertog, Lian Wang, et al.
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