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The Biochemical Journal|January 22, 2015
Role of Cys³⁶⁰² in the function and regulation of the cardiac ryanodine receptorTao Mi, Zhichao Xiao, Wenting Guo, et al.
Journal of Neurogenetics|February 26, 2026
Neurocardiogenetics: Exploring the association of rare <i>RYR2</i> variants with neuropsychiatric disorders in general and disease populationsSaif F Dababneh, Jason D Roberts, Sixuan Tan, et al.
JAMA Cardiology|May 27, 2026
Clinical Diagnosis of Calcium Release Deficiency Syndrome in a Family With Sudden Cardiac DeathJulia C Isbister, Shanshan Tian, Ruiwu Wang, et al.
The Journal of Biological Chemistry|December 9, 2016
The Arrhythmogenic Calmodulin p.Phe142Leu Mutation Impairs C-domain Ca2+ Binding but Not Calmodulin-dependent Inhibition of the Cardiac Ryanodine ReceptorMads Toft Søndergaard, Yingjie Liu, Kamilla Taunsig Larsen, et al.
Mycologia|October 15, 2010
Colletotrichum gloeosporioides s.l. associated with Theobroma cacao and other plants in Panama: multilocus phylogenies distinguish host-associated pathogens from asymptomatic endophytesEnith I Rojas, Stephen A Rehner, Gary J Samuels, et al.
The Journal of Biological Chemistry|October 9, 2007
Removal of FKBP12.6 does not alter the conductance and activation of the cardiac ryanodine receptor or the susceptibility to stress-induced ventricular arrhythmiasJianmin Xiao, Xixi Tian, Peter P Jones, et al.
Bioscience Reports|April 7, 2021
Identification of loss-of-function RyR2 mutations associated with idiopathic ventricular fibrillation and sudden deathXiaowei Zhong, Wenting Guo, Jinhong Wei, et al.
Circulation Research|December 30, 2020
Ca<sup>2+</sup>-CaM Dependent Inactivation of RyR2 Underlies Ca<sup>2+</sup> Alternans in Intact HeartJinhong Wei, Jinjing Yao, Darrell Belke, et al.
The Biochemical Journal|September 2, 2016
Suppression of ryanodine receptor function prolongs Ca2+ release refractoriness and promotes cardiac alternans in intact heartsXiaowei Zhong, Bo Sun, Alexander Vallmitjana, et al.
The Biochemical Journal|July 26, 2023
RyR2 C-terminal truncating variants identified in patients with arrhythmic phenotypes exert a dominant negative effect through formation of wildtype-truncation heteromersShanshan Tian, Xiaowei Zhong, Hui Wang, et al.
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