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Frontiers in Bioscience : a Journal and Virtual Library|February 24, 2004
Chemokines and chemokine receptors in autoimmune encephalomyelitis as a model for central nervous system inflammatory disease regulationRukiye-Nazan E Dogan, William J KarpusJournal of Immunology (Baltimore, Md. : 1950)|May 21, 2008
Production of CCL2 by central nervous system cells regulates development of murine experimental autoimmune encephalomyelitis through the recruitment of TNF- and iNOS-expressing macrophages and myeloid dendritic cellsRukiye-Nazan E Dogan, Adam Elhofy, William J KarpusJournal of Neuroimmunology|May 18, 2011
CCR4 contributes to the pathogenesis of experimental autoimmune encephalomyelitis by regulating inflammatory macrophage functionEileen A Forde, Rukiye-Nazan E Dogan, William J KarpusJournal of Immunology (Baltimore, Md. : 1950)|February 8, 2003
Absence of IL-4, and not suppression of the Th2 response, prevents development of experimental autoimmune Graves' diseaseRukiye-Nazan E Dogan, Chenthamarakshan Vasu, Mark J Holterman, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 22, 2003
Selective induction of dendritic cells using granulocyte macrophage-colony stimulating factor, but not fms-like tyrosine kinase receptor 3-ligand, activates thyroglobulin-specific CD4+/CD25+ T cells and suppresses experimental autoimmune thyroiditisChenthamarakshan Vasu, Rukiye-Nazan E Dogan, Mark J Holterman, et al.Journal of Leukocyte Biology|October 14, 2010
CCL22 regulates experimental autoimmune encephalomyelitis by controlling inflammatory macrophage accumulation and effector functionRukiye-Nazan E Dogan, Nancy Long, Eileen Forde, et al.Pageof 1