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Immunity|December 16, 2014
Myeloid-derived suppressor activity is mediated by monocytic lineages maintained by continuous inhibition of extrinsic and intrinsic death pathwaysJessica M Haverkamp, Amber M Smith, Ricardo Weinlich, et al.
Diabetes|May 22, 2016
CRISPR-Cas9-Mediated Modification of the NOD Mouse Genome With Ptpn22R619W Mutation Increases Autoimmune DiabetesXiaotian Lin, Stephane Pelletier, Sebastien Gingras, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 3, 2013
Two independent pathways of regulated necrosis mediate ischemia-reperfusion injuryAndreas Linkermann, Jan Hinrich Bräsen, Maurice Darding, et al.
Molecular and Cellular Biology|January 26, 2011
MK-STYX, a catalytically inactive phosphatase regulating mitochondrially dependent apoptosisNatalie M Niemi, Nathan J Lanning, Jeff A Klomp, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 13, 2012
Cutting edge: FAS (CD95) mediates noncanonical IL-1β and IL-18 maturation via caspase-8 in an RIP3-independent mannerLukas Bossaller, Ping-I Chiang, Christian Schmidt-Lauber, et al.
Nature Communications|June 25, 2015
Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3Seokwon Kang, Teresa Fernandes-Alnemri, Corey Rogers, et al.
Immunity|August 16, 2016
The Pseudokinase MLKL and the Kinase RIPK3 Have Distinct Roles in Autoimmune Disease Caused by Loss of Death-Receptor-Induced ApoptosisSilvia Alvarez-Diaz, Christopher P Dillon, Najoua Lalaoui, et al.
Molecular Cell|June 11, 2011
Phosphorylation of Tip60 by GSK-3 determines the induction of PUMA and apoptosis by p53Céline Charvet, Manuela Wissler, Prisca Brauns-Schubert, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 27, 2014
Caspase-8 modulates dectin-1 and complement receptor 3-driven IL-1β production in response to β-glucans and the fungal pathogen, Candida albicansSandhya Ganesan, Vijay A K Rathinam, Lukas Bossaller, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 27, 2013
Autosis is a Na+,K+-ATPase-regulated form of cell death triggered by autophagy-inducing peptides, starvation, and hypoxia-ischemiaYang Liu, Sanae Shoji-Kawata, Rhea M Sumpter, et al.
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