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American Journal of Physiology. Cell Physiology|November 15, 2014
Human SLC4A11-C functions as a DIDS-stimulatable H⁺(OH⁻) permeation pathway: partial correction of R109H mutant transportLiyo Kao, Rustam Azimov, Natalia Abuladze, et al.Biochemistry|November 18, 2015
Ion Channel Formation by Tau Protein: Implications for Alzheimer's Disease and TauopathiesNirav Patel, Srinivasan Ramachandran, Rustam Azimov, et al.The Journal of Biological Chemistry|January 9, 2015
Interplay between disulfide bonding and N-glycosylation defines SLC4 Na+-coupled transporter extracellular topographyQuansheng Zhu, Liyo Kao, Rustam Azimov, et al.Peptides|July 20, 2002
The channel hypothesis of Alzheimer's disease: current statusBruce L Kagan, Yutaka Hirakura, Rustam Azimov, et al.The Journal of Biological Chemistry|January 31, 2013
Topology of NBCe1 protein transmembrane segment 1 and structural effect of proximal renal tubular acidosis (pRTA) S427L mutationQuansheng Zhu, Weixin Liu, Liyo Kao, et al.The Journal of Biological Chemistry|January 23, 2009
NBCe1-A Transmembrane Segment 1 Lines the Ion Translocation PathwayQuansheng Zhu, Rustam Azimov, Liyo Kao, et al.The Journal of Physiology|April 9, 2005
Critical amino acid residues involved in the electrogenic sodium-bicarbonate cotransporter kNBC1-mediated transportNatalia Abuladze, Rustam Azimov, Debra Newman, et al.The Journal of Biological Chemistry|March 4, 2010
Topological location and structural importance of the NBCe1-A residues mutated in proximal renal tubular acidosisQuansheng Zhu, Liyo Kao, Rustam Azimov, et al.Nanomedicine : Nanotechnology, Biology, and Medicine|June 13, 2020
Ion channel formation by N-terminally truncated Aβ (4-42): relevance for the pathogenesis of Alzheimer's diseaseAbhijith G Karkisaval, Agueda Rostagno, Rustam Azimov, et al.The Journal of Biological Chemistry|September 15, 2010
Structural and functional characterization of the C-terminal transmembrane region of NBCe1-AQuansheng Zhu, Liyo Kao, Rustam Azimov, et al.Pageof 3