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Diabetes
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November 1, 1988
Exogenous arachidonic acid promotes insulin release from intact or permeabilized rat islets by dual mechanisms. Putative activation of Ca2+ mobilization and protein kinase C
S A Metz
Advances in Prostaglandin, Thromboxane, and Leukotriene Research
|
January 1, 1987
Pancreatic islet phospholipase A2: differential, Ca2+-dependent effects of lysophospholipids, arachidonic acid, and its lipoxygenase-derived metabolites on insulin release
S A Metz
Proceedings of the National Academy of Sciences of the United States of America
|
January 1, 1985
Glucose increases the synthesis of lipoxygenase-mediated metabolites of arachidonic acid in intact rat islets
S A Metz
The Journal of Pharmacology and Experimental Therapeutics
|
September 1, 1986
Putative roles for lysophospholipids as mediators and lipoxygenase-mediated metabolites of arachidonic acid as potentiators of stimulus-secretion coupling: dual mechanisms of p-hydroxymercuribenzoic acid-induced insulin release
S A Metz
Biochemical Pharmacology
|
June 1, 1989
Blockade by lipoxygenase inhibitors of Ca2+-dependent insulin secretion from permeabilized rat islets. A molecular mechanism distinct from that of alpha 2-adrenergic agonists
S A Metz
Diabetes
|
January 1, 1988
Epinephrine impairs insulin release by a mechanism distal to calcium mobilization. Similarity to lipoxygenase inhibitors
S A Metz
Diabetes
|
December 1, 1991
The pancreatic islet as Rubik's Cube. Is phospholipid hydrolysis a piece of the puzzle?
S A Metz
Life Sciences
|
June 9, 1986
A re-interpretation of lipoxygenase-dependent insulin release: which metabolites of arachidonic acid, or none?
S A Metz
JAMA
|
April 23, 1973
Drug treatment of inoperable adrenocortical carcinoma
S A Metz
Prostaglandins
|
January 1, 1984
Is phospholipase A2 a "glucose sensor" responsible for the phasic pattern of insulin release?
S A Metz
Page
of 12
Search research articles
Search
Showing results (11-20 of 114) with videos related to
Sort By:
Page
of 12
Diabetes
|
November 1, 1988
Exogenous arachidonic acid promotes insulin release from intact or permeabilized rat islets by dual mechanisms. Putative activation of Ca2+ mobilization and protein kinase C
S A Metz
Advances in Prostaglandin, Thromboxane, and Leukotriene Research
|
January 1, 1987
Pancreatic islet phospholipase A2: differential, Ca2+-dependent effects of lysophospholipids, arachidonic acid, and its lipoxygenase-derived metabolites on insulin release
S A Metz
Proceedings of the National Academy of Sciences of the United States of America
|
January 1, 1985
Glucose increases the synthesis of lipoxygenase-mediated metabolites of arachidonic acid in intact rat islets
S A Metz
The Journal of Pharmacology and Experimental Therapeutics
|
September 1, 1986
Putative roles for lysophospholipids as mediators and lipoxygenase-mediated metabolites of arachidonic acid as potentiators of stimulus-secretion coupling: dual mechanisms of p-hydroxymercuribenzoic acid-induced insulin release
S A Metz
Biochemical Pharmacology
|
June 1, 1989
Blockade by lipoxygenase inhibitors of Ca2+-dependent insulin secretion from permeabilized rat islets. A molecular mechanism distinct from that of alpha 2-adrenergic agonists
S A Metz
Diabetes
|
January 1, 1988
Epinephrine impairs insulin release by a mechanism distal to calcium mobilization. Similarity to lipoxygenase inhibitors
S A Metz
Diabetes
|
December 1, 1991
The pancreatic islet as Rubik's Cube. Is phospholipid hydrolysis a piece of the puzzle?
S A Metz
Life Sciences
|
June 9, 1986
A re-interpretation of lipoxygenase-dependent insulin release: which metabolites of arachidonic acid, or none?
S A Metz
JAMA
|
April 23, 1973
Drug treatment of inoperable adrenocortical carcinoma
S A Metz
Prostaglandins
|
January 1, 1984
Is phospholipase A2 a "glucose sensor" responsible for the phasic pattern of insulin release?
S A Metz
Page
of 12