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Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|June 16, 2025
Association of Homologous Recombination Repair Alterations With Outcomes in Patients with Metastatic Hormone-Sensitive Prostate CancerChadi Hage Chehade, Ryon P Graf, Zeynep Irem Ozay, et al.
The Prostate|September 23, 2024
Bipolar androgen therapy for treatment of metastatic castration-resistant prostate cancer: A case seriesElizabeth U Tran, Eric Royz, Kyra Yamamoto, et al.
Urology Case Reports|March 16, 2018
Diagnosing small bowel carcinoid tumor in a patient with oligometastatic prostate cancer imaged with PSMA-Targeted [18F]DCFPyL PET/CT: Value of the PSMA-RADS-3D DesignationEugene Shenderov, Michael A Gorin, Seohyun Kim, et al.
Cancer|June 8, 2012
An immunohistochemical signature comprising PTEN, MYC, and Ki67 predicts progression in prostate cancer patients receiving adjuvant docetaxel after prostatectomyEmmanuel S Antonarakis, Daniel Keizman, Zhe Zhang, et al.
Prostate Cancer and Prostatic Diseases|November 6, 2013
Very-high-risk localized prostate cancer: definition and outcomesD Sundi, V M Wang, P M Pierorazio, et al.
Journal of the National Cancer Institute|March 8, 2020
Putting the Pieces Together: Completing the Mechanism of Action Jigsaw for Sipuleucel-TRavi A Madan, Emmanuel S Antonarakis, Charles G Drake, et al.
European Urology Oncology|October 8, 2023
Clinical and Functional Analyses of an African-ancestry Gain-of-function HOXB13 Variant Implicated in Aggressive Prostate CancerMayuko Kanayama, Yidong Chen, Daniel Rabizadeh, et al.
Cancer Immunology Research|June 7, 2015
A Paracrine Role for IL6 in Prostate Cancer Patients: Lack of Production by Primary or Metastatic Tumor CellsShu-Han Yu, Qizhi Zheng, David Esopi, et al.
The Oncologist|July 12, 2019
Pan-Cancer Analysis of CDK12 Loss-of-Function Alterations and Their Association with the Focal Tandem-Duplicator PhenotypeEthan S Sokol, Dean Pavlick, Garrett M Frampton, et al.
Genome Research|December 16, 2000
The mouse brain transcriptome by SAGE: differences in gene expression between P30 brains of the partial trisomy 16 mouse model of Down syndrome (Ts65Dn) and normalsR Chrast, H S Scott, M P Papasavvas, et al.
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