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S B Dalton

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AIDS Research and Human Retroviruses|August 25, 2000
Simian-human immunodeficiency virus-associated nephropathy in macaquesE B Stephens, C Tian, S B Dalton, et al.
AIDS Research and Human Retroviruses|August 10, 2000
Comparison of Vpu sequences from diverse geographical isolates of HIV type 1 identifies the presence of highly variable domains, additional invariant amino acids, and a signature sequence motif common to subtype C isolatesC McCormick-Davis, S B Dalton, D K Singh, et al.
Virology|June 30, 2000
A molecular clone of simian-human immunodeficiency virus (DeltavpuSHIV(KU-1bMC33)) with a truncated, non-membrane-bound vpu results in rapid CD4(+) T cell loss and neuro-AIDS in pig-tailed macaquesC McCormick-Davis, S B Dalton, D R Hout, et al.
Virology|March 22, 2001
A simian human immunodeficiency virus with a nonfunctional Vpu (deltavpuSHIV(KU-1bMC33)) isolated from a macaque with neuroAIDS has selected for mutations in env and nef that contributed to its pathogenic phenotypeD K Singh, C McCormick, E Pacyniak, et al.
Pageof 1

Showing results (1-10 of 4) with videos related to

Sort By:
Pageof 1
AIDS Research and Human Retroviruses|August 25, 2000
Simian-human immunodeficiency virus-associated nephropathy in macaquesE B Stephens, C Tian, S B Dalton, et al.
AIDS Research and Human Retroviruses|August 10, 2000
Comparison of Vpu sequences from diverse geographical isolates of HIV type 1 identifies the presence of highly variable domains, additional invariant amino acids, and a signature sequence motif common to subtype C isolatesC McCormick-Davis, S B Dalton, D K Singh, et al.
Virology|June 30, 2000
A molecular clone of simian-human immunodeficiency virus (DeltavpuSHIV(KU-1bMC33)) with a truncated, non-membrane-bound vpu results in rapid CD4(+) T cell loss and neuro-AIDS in pig-tailed macaquesC McCormick-Davis, S B Dalton, D R Hout, et al.
Virology|March 22, 2001
A simian human immunodeficiency virus with a nonfunctional Vpu (deltavpuSHIV(KU-1bMC33)) isolated from a macaque with neuroAIDS has selected for mutations in env and nef that contributed to its pathogenic phenotypeD K Singh, C McCormick, E Pacyniak, et al.
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