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S C Bock

Showing results (21-30 of 33) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|October 1, 1985
Human apolipoprotein B cDNA clone isolation and demonstration that liver apolipoprotein B mRNA is 22 kilobases in lengthL S Huang, S C Bock, S I Feinstein, et al.
Science (New York, N.Y.)|January 10, 1986
Modification of the active site of alkaline phosphatase by site-directed mutagenesisS S Ghosh, S C Bock, S E Rokita, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 1, 1982
Hybridization-selected translation of Bombyx mori high-cysteine chorion proteins in Xenopus laevis oocytesS C Bock, D C Tiemeier, K Mester, et al.
The Journal of Biological Chemistry|February 25, 1989
CpG mutations in the reactive site of human C1 inhibitorK Skriver, E Radziejewska, J A Silbermann, et al.
Biochemistry|May 31, 2001
The antithrombin P1 residue is important for target proteinase specificity but not for heparin activation of the serpin. Characterization of P1 antithrombin variants with altered proteinase specificity but normal heparin activationY J Chuang, R Swanson, S M Raja, et al.
Analytical Biochemistry|October 15, 1979
Two-dimensional electrophoresis of small-molecular-weight proteinsM R Goldsmith, E C Rattner, M M Koehler, et al.
FEBS Letters|April 6, 1992
Antithrombin Budapest 3. An antithrombin variant with reduced heparin affinity resulting from the substitution L99FR J Olds, D A Lane, M Boisclair, et al.
The Journal of Biological Chemistry|May 15, 1991
Substrate properties of C1 inhibitor Ma (alanine 434----glutamic acid). Genetic and structural evidence suggesting that the P12-region contains critical determinants of serine protease inhibitor/substrate statusK Skriver, W R Wikoff, P A Patston, et al.
American Journal of Human Genetics|January 1, 1985
Hereditary thrombosis in a Utah kindred is caused by a dysfunctional antithrombin III geneS C Bock, J F Harris, C E Schwartz, et al.
The Biochemical Journal|August 15, 1995
Elimination of glycosylation heterogeneity affecting heparin affinity of recombinant human antithrombin III by expression of a beta-like variant in baculovirus-infected insect cellsE Ersdal-Badju, A Lu, X Peng, et al.
Pageof 4

Showing results (21-30 of 33) with videos related to

Sort By:
Pageof 4
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1985
Human apolipoprotein B cDNA clone isolation and demonstration that liver apolipoprotein B mRNA is 22 kilobases in lengthL S Huang, S C Bock, S I Feinstein, et al.
Science (New York, N.Y.)|January 10, 1986
Modification of the active site of alkaline phosphatase by site-directed mutagenesisS S Ghosh, S C Bock, S E Rokita, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 1, 1982
Hybridization-selected translation of Bombyx mori high-cysteine chorion proteins in Xenopus laevis oocytesS C Bock, D C Tiemeier, K Mester, et al.
The Journal of Biological Chemistry|February 25, 1989
CpG mutations in the reactive site of human C1 inhibitorK Skriver, E Radziejewska, J A Silbermann, et al.
Biochemistry|May 31, 2001
The antithrombin P1 residue is important for target proteinase specificity but not for heparin activation of the serpin. Characterization of P1 antithrombin variants with altered proteinase specificity but normal heparin activationY J Chuang, R Swanson, S M Raja, et al.
Analytical Biochemistry|October 15, 1979
Two-dimensional electrophoresis of small-molecular-weight proteinsM R Goldsmith, E C Rattner, M M Koehler, et al.
FEBS Letters|April 6, 1992
Antithrombin Budapest 3. An antithrombin variant with reduced heparin affinity resulting from the substitution L99FR J Olds, D A Lane, M Boisclair, et al.
The Journal of Biological Chemistry|May 15, 1991
Substrate properties of C1 inhibitor Ma (alanine 434----glutamic acid). Genetic and structural evidence suggesting that the P12-region contains critical determinants of serine protease inhibitor/substrate statusK Skriver, W R Wikoff, P A Patston, et al.
American Journal of Human Genetics|January 1, 1985
Hereditary thrombosis in a Utah kindred is caused by a dysfunctional antithrombin III geneS C Bock, J F Harris, C E Schwartz, et al.
The Biochemical Journal|August 15, 1995
Elimination of glycosylation heterogeneity affecting heparin affinity of recombinant human antithrombin III by expression of a beta-like variant in baculovirus-infected insect cellsE Ersdal-Badju, A Lu, X Peng, et al.
Pageof 4