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Cell|October 21, 1994
DNA damage can induce apoptosis in proliferating lymphoid cells via p53-independent mechanisms inhibitable by Bcl-2A Strasser, A W Harris, T Jacks, et al.Nature|March 31, 1994
Bcl-2 expression promotes B- but not T-lymphoid development in scid miceA Strasser, A W Harris, L M Corcoran, et al.Oncogene|September 28, 1999
Transgenic models of lymphoid neoplasia and development of a pan-hematopoietic vectorJ M Adams, A W Harris, A Strasser, et al.Cancer Research|April 10, 1999
Insights from Bcl-2 and Myc: malignancy involves abrogation of apoptosis as well as sustained proliferationS Cory, D L Vaux, A Strasser, et al.Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|August 30, 1994
Insights from transgenic mice regarding the role of bcl-2 in normal and neoplastic lymphoid cellsS Cory, A W Harris, A StrasserCell|January 1, 1985
Transposition of the immunoglobulin heavy chain enhancer to the myc oncogene in a murine plasmacytomaL M Corcoran, S Cory, J M AdamsCell|November 29, 1991
bcl-2 transgene inhibits T cell death and perturbs thymic self-censorshipA Strasser, A W Harris, S CoryOncogene|January 1, 1993
E mu-bcl-2 transgene facilitates spontaneous transformation of early pre-B and immunoglobulin-secreting cells but not T cellsA Strasser, A W Harris, S CoryCell|July 14, 1995
Absence of p53 allows direct immortalization of hematopoietic cells by the myc and raf oncogenesT Metz, A W Harris, J M AdamsProceedings of the National Academy of Sciences of the United States of America|October 1, 1991
Enforced BCL2 expression in B-lymphoid cells prolongs antibody responses and elicits autoimmune diseaseA Strasser, S Whittingham, D L Vaux, et al.Pageof 89