Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

S Dimmeler

Showing results (21-30 of 87) with videos related to

Pageof 9
Sort By:
FEBS Letters|March 30, 2001
Pro-atherogenic factors induce telomerase inactivation in endothelial cells through an Akt-dependent mechanismK Breitschopf, A M Zeiher, S Dimmeler
FEBS Letters|July 26, 2000
Phosphorylation of the endothelial nitric oxide synthase at ser-1177 is required for VEGF-induced endothelial cell migrationS Dimmeler, E Dernbach, A M Zeiher
The Journal of Biological Chemistry|May 10, 2000
Ubiquitin-mediated degradation of the proapoptotic active form of bid. A functional consequence on apoptosis inductionK Breitschopf, A M Zeiher, S Dimmeler
European Journal of Pharmacology|December 19, 1996
Vitamin C and E prevent lipopolysaccharide-induced apoptosis in human endothelial cells by modulation of Bcl-2 and BaxJ Haendeler, A M Zeiher, S Dimmeler
Basic Research in Cardiology|February 24, 2001
Apoptosis in the vascular wall and atherosclerosisL Rössig, S Dimmeler, A M Zeiher
Journal of Immunology (Baltimore, Md. : 1950)|April 1, 1993
Exogenous nitric oxide (NO) generation or IL-1 beta-induced intracellular NO production stimulates inhibitory auto-ADP-ribosylation of glyceraldehyde-3-phosphate dehydrogenase in RINm5F cellsS Dimmeler, M Ankarcrona, P Nicotera, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 12, 1999
Upregulation of superoxide dismutase and nitric oxide synthase mediates the apoptosis-suppressive effects of shear stress on endothelial cellsS Dimmeler, C Hermann, J Galle, et al.
The Journal of Experimental Medicine|June 8, 1999
Dephosphorylation targets Bcl-2 for ubiquitin-dependent degradation: a link between the apoptosome and the proteasome pathwayS Dimmeler, K Breitschopf, J Haendeler, et al.
Deutsche Medizinische Wochenschrift (1946)|February 23, 1996
[Mechanisms of action of high-dosage glucocorticoid therapy]F Buttgereit, S Dimmeler, E Neugebauer, et al.
Circulation|April 1, 1997
Oxidized low-density lipoprotein induces apoptosis of human endothelial cells by activation of CPP32-like proteases. A mechanistic clue to the 'response to injury' hypothesisS Dimmeler, J Haendeler, J Galle, et al.
Pageof 9

Showing results (21-30 of 87) with videos related to

Sort By:
Pageof 9
FEBS Letters|March 30, 2001
Pro-atherogenic factors induce telomerase inactivation in endothelial cells through an Akt-dependent mechanismK Breitschopf, A M Zeiher, S Dimmeler
FEBS Letters|July 26, 2000
Phosphorylation of the endothelial nitric oxide synthase at ser-1177 is required for VEGF-induced endothelial cell migrationS Dimmeler, E Dernbach, A M Zeiher
The Journal of Biological Chemistry|May 10, 2000
Ubiquitin-mediated degradation of the proapoptotic active form of bid. A functional consequence on apoptosis inductionK Breitschopf, A M Zeiher, S Dimmeler
European Journal of Pharmacology|December 19, 1996
Vitamin C and E prevent lipopolysaccharide-induced apoptosis in human endothelial cells by modulation of Bcl-2 and BaxJ Haendeler, A M Zeiher, S Dimmeler
Basic Research in Cardiology|February 24, 2001
Apoptosis in the vascular wall and atherosclerosisL Rössig, S Dimmeler, A M Zeiher
Journal of Immunology (Baltimore, Md. : 1950)|April 1, 1993
Exogenous nitric oxide (NO) generation or IL-1 beta-induced intracellular NO production stimulates inhibitory auto-ADP-ribosylation of glyceraldehyde-3-phosphate dehydrogenase in RINm5F cellsS Dimmeler, M Ankarcrona, P Nicotera, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 12, 1999
Upregulation of superoxide dismutase and nitric oxide synthase mediates the apoptosis-suppressive effects of shear stress on endothelial cellsS Dimmeler, C Hermann, J Galle, et al.
The Journal of Experimental Medicine|June 8, 1999
Dephosphorylation targets Bcl-2 for ubiquitin-dependent degradation: a link between the apoptosome and the proteasome pathwayS Dimmeler, K Breitschopf, J Haendeler, et al.
Deutsche Medizinische Wochenschrift (1946)|February 23, 1996
[Mechanisms of action of high-dosage glucocorticoid therapy]F Buttgereit, S Dimmeler, E Neugebauer, et al.
Circulation|April 1, 1997
Oxidized low-density lipoprotein induces apoptosis of human endothelial cells by activation of CPP32-like proteases. A mechanistic clue to the 'response to injury' hypothesisS Dimmeler, J Haendeler, J Galle, et al.
Pageof 9