Showing results (151-160 of 208) with videos related to

Sort By:
Pageof 21
The Journal of Clinical Endocrinology and Metabolism|April 1, 1982
Progressive adrenal failure in polyglandular autoimmune diseaseP Saenger, L S Levine, W J Irvine, et al.
Life Sciences|May 4, 1987
Diet-hormone interactions: protein/carbohydrate ratio alters reciprocally the plasma levels of testosterone and cortisol and their respective binding globulins in manK E Anderson, W Rosner, M S Khan, et al.
The Journal of Clinical Endocrinology and Metabolism|September 13, 2003
Congenital hypopituitarism as a cause of undetectable estriol levels in the maternal triple-marker screenI Marshall, F Ugrasbul, F Manginello, et al.
The Journal of Pediatrics|February 1, 1980
Depressed serum somatomedin activity in beta-thalassemiaP Saenger, E Schwartz, A L Markenson, et al.
Molecular and Cellular Endocrinology|May 17, 1996
Point mutations abolish 11 beta-hydroxysteroid dehydrogenase type II activity in three families with the congenital syndrome of apparent mineralocorticoid excessP Ferrari, V R Obeyesekere, K Li, et al.
The Journal of Clinical Endocrinology and Metabolism|July 1, 1979
Adrenocortical function, electrolyte metabolism, and blood pressure during prolonged adrenocorticotropin infusion in juvenile hypertensionW Rauh, L S Levine, K Gottesdiener, et al.
Padiatrie Und Padologie. Supplementum|January 1, 1977
Somatomedin and growth hormone in psychosocial dwarfismP Saenger, L S Levine, E Wiedemann, et al.
The Journal of Clinical Endocrinology and Metabolism|March 1, 1985
Late-onset adrenal steroid 3 beta-hydroxysteroid dehydrogenase deficiency. I. A cause of hirsutism in pubertal and postpubertal womenS Y Pang, A J Lerner, E Stoner, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 1, 1985
Two genes encoding steroid 21-hydroxylase are located near the genes encoding the fourth component of complement in manP C White, D Grossberger, B J Onufer, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 1, 1992
Glucocorticoid-suppressible hyperaldosteronism results from hybrid genes created by unequal crossovers between CYP11B1 and CYP11B2L Pascoe, K M Curnow, L Slutsker, et al.
Pageof 21