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The Journal of Clinical Endocrinology and Metabolism|December 1, 1992
Insulin insensitivity in adrenal hyperplasia due to nonclassical steroid 21-hydroxylase deficiencyP W Speiser, J Serrat, M I New, et al.American Journal of Human Genetics|October 1, 1986
Frequent deletion and duplication of the steroid 21-hydroxylase genesJ W Werkmeister, M I New, B Dupont, et al.Human Genetics|January 1, 1981
The biochemical basis for genotyping 21-hydroxylase deficiencyM I New, B Dupont, M S Pollack, et al.Metabolism: Clinical and Experimental|July 1, 1993
Investigation of the mechanism of hypertension in apparent mineralocorticoid excessP W Speiser, L M Riddick, K Martin, et al.The New England Journal of Medicine|May 21, 1987
Hirsutism, polycystic ovarian disease, and ovarian 17-ketosteroid reductase deficiencyS Y Pang, B Softness, W J Sweeney, et al.Prenatal Diagnosis|March 4, 1998
Normal external genitalia in a female with classical congenital adrenal hyperplasia who was not treated during embryogenesisN Quercia, D Chitayat, R Babul-Hirji, et al.Psychoneuroendocrinology|January 1, 1995
Effects of early prenatal dexamethasone on the cognitive and behavioral development of young children: results of a pilot studyP D Trautman, H F Meyer-Bahlburg, J Postelnek, et al.The Journal of Clinical Endocrinology and Metabolism|August 1, 1985
Excess mineralocorticoid receptor activity in patients with dexamethasone-suppressible hyperaldosteronism is under adrenocorticotropin controlP W Speiser, K O Martin, G Kao-Lo, et al.The Journal of Clinical Endocrinology and Metabolism|December 22, 1999
Prevalence of mild apparent mineralocorticoid excess in MennonitesF Ugrasbul, T Wiens, P Rubinstein, et al.Pageof 21