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S Duckett

Showing results (161-170 of 194) with videos related to

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The EMBO Journal|September 1, 1993
A cooperative interaction between NF-kappa B and Sp1 is required for HIV-1 enhancer activationN D Perkins, N L Edwards, C S Duckett, et al.
ASAIO Transactions|July 1, 1986
Transmural blood flow of multi-layered latissimus dorsi skeletal muscle ventricles during circulatory assistanceJ D Mannion, M A Velchik, M Acker, et al.
The Biochemical Journal|May 16, 2003
Identification of ubiquitination sites on the X-linked inhibitor of apoptosis proteinHwain Shin, Kazuya Okada, John C Wilkinson, et al.
European Journal of Immunology|November 28, 2000
Patients with X-linked lymphoproliferative disease have a defect in 2B4 receptor-mediated NK cell cytotoxicityH Nakajima, M Cella, A Bouchon, et al.
The Biochemical Journal|February 16, 2012
An inactivating caspase 11 passenger mutation originating from the 129 murine strain in mice targeted for c-IAP1Niall S Kenneth, J Michael Younger, Elizabeth D Hughes, et al.
The Journal of Biological Chemistry|September 17, 2004
VIAF, a conserved inhibitor of apoptosis (IAP)-interacting factor that modulates caspase activationJohn C Wilkinson, Bettina W M Richter, Amanda S Wilkinson, et al.
Breast Cancer Research and Treatment|April 25, 2007
EZH2 regulates the transcription of estrogen-responsive genes through association with REA, an estrogen receptor corepressorClara Hwang, Veda N Giri, John C Wilkinson, et al.
The Journal of Biological Chemistry|September 22, 2001
SNIP1 inhibits NF-kappa B signaling by competing for its binding to the C/H1 domain of CBP/p300 transcriptional co-activatorsR H Kim, K C Flanders, S Birkey Reffey, et al.
The Journal of Biological Chemistry|December 7, 2000
c-IAP1 is cleaved by caspases to produce a proapoptotic C-terminal fragmentR J Clem, T T Sheu, B W Richter, et al.
Molecular and Cellular Biology|March 1, 1993
Dimerization of NF-KB2 with RelA(p65) regulates DNA binding, transcriptional activation, and inhibition by an I kappa B-alpha (MAD-3)C S Duckett, N D Perkins, T F Kowalik, et al.
Pageof 20

Showing results (161-170 of 194) with videos related to

Sort By:
Pageof 20
The EMBO Journal|September 1, 1993
A cooperative interaction between NF-kappa B and Sp1 is required for HIV-1 enhancer activationN D Perkins, N L Edwards, C S Duckett, et al.
ASAIO Transactions|July 1, 1986
Transmural blood flow of multi-layered latissimus dorsi skeletal muscle ventricles during circulatory assistanceJ D Mannion, M A Velchik, M Acker, et al.
The Biochemical Journal|May 16, 2003
Identification of ubiquitination sites on the X-linked inhibitor of apoptosis proteinHwain Shin, Kazuya Okada, John C Wilkinson, et al.
European Journal of Immunology|November 28, 2000
Patients with X-linked lymphoproliferative disease have a defect in 2B4 receptor-mediated NK cell cytotoxicityH Nakajima, M Cella, A Bouchon, et al.
The Biochemical Journal|February 16, 2012
An inactivating caspase 11 passenger mutation originating from the 129 murine strain in mice targeted for c-IAP1Niall S Kenneth, J Michael Younger, Elizabeth D Hughes, et al.
The Journal of Biological Chemistry|September 17, 2004
VIAF, a conserved inhibitor of apoptosis (IAP)-interacting factor that modulates caspase activationJohn C Wilkinson, Bettina W M Richter, Amanda S Wilkinson, et al.
Breast Cancer Research and Treatment|April 25, 2007
EZH2 regulates the transcription of estrogen-responsive genes through association with REA, an estrogen receptor corepressorClara Hwang, Veda N Giri, John C Wilkinson, et al.
The Journal of Biological Chemistry|September 22, 2001
SNIP1 inhibits NF-kappa B signaling by competing for its binding to the C/H1 domain of CBP/p300 transcriptional co-activatorsR H Kim, K C Flanders, S Birkey Reffey, et al.
The Journal of Biological Chemistry|December 7, 2000
c-IAP1 is cleaved by caspases to produce a proapoptotic C-terminal fragmentR J Clem, T T Sheu, B W Richter, et al.
Molecular and Cellular Biology|March 1, 1993
Dimerization of NF-KB2 with RelA(p65) regulates DNA binding, transcriptional activation, and inhibition by an I kappa B-alpha (MAD-3)C S Duckett, N D Perkins, T F Kowalik, et al.
Pageof 20