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S E Andrew

Showing results (31-40 of 53) with videos related to

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Mutation Research|October 25, 1996
A novel lacI transgenic mutation-detection system and its application to establish baseline mutation frequencies in the scid mouseS E Andrew, S Pownall, J Fox, et al.
Journal of Medical Genetics|December 1, 1993
Familial predisposition to recurrent mutations causing Huntington's disease: genetic risk to sibs of sporadic casesY P Goldberg, S E Andrew, J Theilmann, et al.
Oncogene|July 10, 1997
Base transitions dominate the mutational spectrum of a transgenic reporter gene in MSH2 deficient miceS E Andrew, A H Reitmair, J Fox, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 14, 1998
Tissues of MSH2-deficient mice demonstrate hypermutability on exposure to a DNA methylating agentS E Andrew, M McKinnon, B S Cheng, et al.
Journal of the American Veterinary Medical Association|June 9, 2000
Development of glaucoma after cataract surgery in dogs: 220 cases (1987-1998)D J Biros, K N Gelatt, D E Brooks, et al.
The New England Journal of Medicine|May 19, 1994
A worldwide study of the Huntington's disease mutation. The sensitivity and specificity of measuring CAG repeatsB Kremer, P Goldberg, S E Andrew, et al.
American Journal of Human Genetics|May 1, 1994
Huntington disease without CAG expansion: phenocopies or errors in assignment?S E Andrew, Y P Goldberg, B Kremer, et al.
Human Molecular Genetics|December 1, 1994
DNA haplotype analysis of Huntington disease reveals clues to the origins and mechanisms of CAG expansion and reasons for geographic variations of prevalenceF Squitieri, S E Andrew, Y P Goldberg, et al.
Carcinogenesis|June 30, 2000
Mutagenesis in PMS2- and MSH2-deficient mice indicates differential protection from transversions and frameshiftsS E Andrew, X S Xu, A Baross-Francis, et al.
Human Molecular Genetics|October 1, 1993
Molecular analysis of juvenile Huntington disease: the major influence on (CAG)n repeat length is the sex of the affected parentH Telenius, H P Kremer, J Theilmann, et al.
Pageof 6

Showing results (31-40 of 53) with videos related to

Sort By:
Pageof 6
Mutation Research|October 25, 1996
A novel lacI transgenic mutation-detection system and its application to establish baseline mutation frequencies in the scid mouseS E Andrew, S Pownall, J Fox, et al.
Journal of Medical Genetics|December 1, 1993
Familial predisposition to recurrent mutations causing Huntington's disease: genetic risk to sibs of sporadic casesY P Goldberg, S E Andrew, J Theilmann, et al.
Oncogene|July 10, 1997
Base transitions dominate the mutational spectrum of a transgenic reporter gene in MSH2 deficient miceS E Andrew, A H Reitmair, J Fox, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 14, 1998
Tissues of MSH2-deficient mice demonstrate hypermutability on exposure to a DNA methylating agentS E Andrew, M McKinnon, B S Cheng, et al.
Journal of the American Veterinary Medical Association|June 9, 2000
Development of glaucoma after cataract surgery in dogs: 220 cases (1987-1998)D J Biros, K N Gelatt, D E Brooks, et al.
The New England Journal of Medicine|May 19, 1994
A worldwide study of the Huntington's disease mutation. The sensitivity and specificity of measuring CAG repeatsB Kremer, P Goldberg, S E Andrew, et al.
American Journal of Human Genetics|May 1, 1994
Huntington disease without CAG expansion: phenocopies or errors in assignment?S E Andrew, Y P Goldberg, B Kremer, et al.
Human Molecular Genetics|December 1, 1994
DNA haplotype analysis of Huntington disease reveals clues to the origins and mechanisms of CAG expansion and reasons for geographic variations of prevalenceF Squitieri, S E Andrew, Y P Goldberg, et al.
Carcinogenesis|June 30, 2000
Mutagenesis in PMS2- and MSH2-deficient mice indicates differential protection from transversions and frameshiftsS E Andrew, X S Xu, A Baross-Francis, et al.
Human Molecular Genetics|October 1, 1993
Molecular analysis of juvenile Huntington disease: the major influence on (CAG)n repeat length is the sex of the affected parentH Telenius, H P Kremer, J Theilmann, et al.
Pageof 6