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The American Journal of Physiology|September 1, 1994
Ca2+ handling and myofibrillar Ca2+ sensitivity in ferret cardiac myocytes with pressure-overload hypertrophyJ Wang, K Flemal, Z Qiu, et al.Circulation|July 1, 1993
Survival after the onset of congestive heart failure in Framingham Heart Study subjectsK K Ho, K M Anderson, W B Kannel, et al.Radiology|March 1, 1981
Film reciprocity law failure in scintillation camera imagingL W Grossman, R J Van Tuinen, J B Kruger, et al.Circulation|March 1, 1982
Modification of abnormal left ventricular diastolic properties by nifedipine in patients with hypertrophic cardiomyopathyB H Lorell, W J Paulus, W Grossman, et al.The American Journal of Physiology|January 1, 1990
Chronic inhibition of fatty acid oxidation: new model of diastolic dysfunctionS E Litwin, T E Raya, R G Gay, et al.Journal of Molecular and Cellular Cardiology|January 1, 1995
The spontaneously hypertensive rat as a model of the transition from compensated left ventricular hypertrophy to failureO H Bing, W W Brooks, K G Robinson, et al.The American Journal of Physiology|November 1, 1996
GH replacement fails to improve ventricular function in hypophysectomized rats with myocardial infarctionY T Shen, R T Wiedmann, J J Lynch, et al.Circulation|February 1, 1990
Abnormal intracellular calcium handling, a major cause of systolic and diastolic dysfunction in ventricular myocardium from patients with heart failureJ P Morgan, R E Erny, P D Allen, et al.Cardiovascular Research|February 1, 1993
Relationship of abnormal intracellular calcium mobilisation to myocyte hypertrophy in human ventricular myocardiumJ K Gwathmey, L A Bentivegna, B J Ransil, et al.The American Journal of Cardiology|December 1, 1979
Myocardial contractile function in aortic stenosis as determined from the rate of stress development during isovolumic systoleM A Fifer, S Gunther, W Grossman, et al.Pageof 28