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Thrombosis and Haemostasis|May 11, 1999
Venous thromboembolic disease and the prothrombin, methylene tetrahydrofolate reductase and factor V genesM Alhenc-Gelas, E Arnaud, V Nicaud, et al.
Arteriosclerosis and Thrombosis : a Journal of Vascular Biology|December 1, 1994
Three novel mutations of antithrombin inducing high-molecular-mass compoundsJ Emmerich, D Vidaud, M Alhenc-Gelas, et al.
Thrombosis and Haemostasis|October 1, 1994
Molecular basis of antithrombin type I deficiency: the first large in-frame deletion and two novel mutations in exon 6J Emmerich, G Chadeuf, M Alhenc-Gelas, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|October 1, 1996
Homozygous variant of antithrombin with lack of affinity for heparin: management of severe thrombotic complications associated with intrauterine fetal demiseA Bauters, C Zawadzki, A Bura, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|February 17, 2000
Protective effect of a thrombin receptor (protease-activated receptor 1) gene polymorphism toward venous thromboembolismE Arnaud, V Nicaud, O Poirier, et al.
Thrombosis and Haemostasis|December 18, 1987
Circulating activities during constant infusion of heparin or a low molecular weight derivative (enoxaparine): failure to demonstrate any circadian variationsP Toulon, J F Vitoux, C Leroy, et al.
Journal of Thrombosis and Haemostasis : JTH|November 20, 2004
Diagnostic score for heparin-induced thrombocytopenia after cardiopulmonary bypassA Lillo-Le Louët, P Boutouyrie, M Alhenc-Gelas, et al.
British Journal of Clinical Pharmacology|December 1, 1995
Comparison of biological activities of two low molecular weight heparins in 10 healthy volunteersM Azizi, C Veyssier-Belot, M Alhenc-Gelas, et al.
La Revue De Medecine Interne|September 1, 1984
[Intra-arterial thrombolysis with the combination of urokinase and lysyl-plasminogen. 27 cases of acute arterial obliteration of the lower limbs]J F Vitoux, J M Pernes, M Roncato, et al.
Blood|January 1, 1995
Identification of 15 different candidate causal point mutations and three polymorphisms in 19 patients with protein S deficiency using a scanning method for the analysis of the protein S active geneS Gandrille, D Borgel, V Eschwege-Gufflet, et al.
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