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Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|February 1, 1995
Protein C infusion in a patient with inherited protein C deficiency caused by two missense mutations: Arg 178 to Gln and Arg-1 to HisM Alhenc-Gelas, J Emmerich, S Gandrille, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|April 9, 1999
Mutations in promoter region of thrombomodulin and venous thromboembolic diseaseL Le Flem, V Picard, J Emmerich, et al.
Thrombosis and Haemostasis|June 16, 1988
A study of fibrinogen and fibrinolysis in 10 adults with nephrotic syndromeS Gandrille, M H Jouvin, P Toulon, et al.
British Journal of Haematology|July 1, 1991
Molecular basis for hereditary antithrombin III quantitative deficiencies: a stop codon in exon IIIa and a frameshift in exon VIS Gandrille, D Vidaud, J Emmerich, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 11, 1999
Complex association of protein C gene promoter polymorphism with circulating protein C levels and thrombotic riskM Aiach, V Nicaud, M Alhenc-Gelas, et al.
British Journal of Haematology|October 16, 1999
Influence of three potential genetic risk factors for thrombosis in 43 families carrying the factor V Arg 506 to Gln mutationV Le Cam-Duchez, S Gandrille, D Trégouët, et al.
Blood|July 1, 1995
Incidence of activated protein C resistance caused by the ARG 506 GLN mutation in factor V in 113 unrelated symptomatic protein C-deficient patients. The French Network on the behalf of INSERMS Gandrille, J S Greengard, M Alhenc-Gelas, et al.
Thrombosis and Haemostasis|March 4, 1999
The factor V gene A4070G mutation and the risk of venous thrombosisM Alhenc-Gelas, V Nicaud, S Gandrille, et al.
Human Mutation|January 1, 1992
Two novel mutations responsible for hereditary type I protein C deficiency: characterization by denaturing gradient gel electrophoresisS Gandrille, M Vidaud, M Aiach, et al.
Blood|November 1, 1995
The Ser 460 to Pro substitution of the protein S alpha (PROS1) gene is a frequent mutation associated with free protein S (type IIa) deficiencyJ Duchemin, S Gandrille, D Borgel, et al.
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