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Journal of Cardiovascular Pharmacology|June 8, 2001
Effects of ACE inhibition on left ventricular failure and oxidative stress in Dahl salt-sensitive ratsH Tsutsui, T Ide, S Hayashidani, et al.Cardiovascular Research|December 21, 2000
Greater susceptibility of failing cardiac myocytes to oxygen free radical-mediated injuryH Tsutsui, T Ide, S Hayashidani, et al.Circulation Research|September 2, 2000
Treatment with dimethylthiourea prevents left ventricular remodeling and failure after experimental myocardial infarction in mice: role of oxidative stressS Kinugawa, H Tsutsui, S Hayashidani, et al.American Journal of Physiology. Heart and Circulatory Physiology|July 17, 2001
Alpha1-adrenoceptor-Gq-RhoA signaling is upregulated to increase myofibrillar Ca2+ sensitivity in failing heartsN Suematsu, S Satoh, S Kinugawa, et al.Circulation Research|March 16, 2001
Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarctionT Ide, H Tsutsui, S Hayashidani, et al.Journal of Cardiovascular Pharmacology|January 11, 2001
Positive inotropic effects of calcium sensitizers on normal and failing cardiac myocytesH Tsutsui, S Kinugawa, T Ide, et al.Circulation|July 12, 2001
Enhanced generation of reactive oxygen species in the limb skeletal muscles from a murine infarct model of heart failureH Tsutsui, T Ide, S Hayashidani, et al.Circulation Research|February 10, 2000
Direct evidence for increased hydroxyl radicals originating from superoxide in the failing myocardiumT Ide, H Tsutsui, S Kinugawa, et al.Circulation|December 12, 2001
8-oxo-dGTPase, which prevents oxidative stress-induced DNA damage, increases in the mitochondria from failing heartsH Tsutsui, T Ide, T Shiomi, et al.Pageof 1