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Showing results (41-50 of 44) with videos related to

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American Journal of Human Genetics|September 3, 2002
A gene mutated in nephronophthisis and retinitis pigmentosa encodes a novel protein, nephroretinin, conserved in evolutionEdgar Otto, Julia Hoefele, Rainer Ruf, et al.
Trials|April 9, 2014
Evaluation of a practice team-supported exposure training for patients with panic disorder with or without agoraphobia in primary care - study protocol of a cluster randomised controlled superiority trialJochen Gensichen, Thomas S Hiller, Jörg Breitbart, et al.
The Journal of Clinical Investigation|September 19, 2001
7-Dehydrocholesterol-dependent proteolysis of HMG-CoA reductase suppresses sterol biosynthesis in a mouse model of Smith-Lemli-Opitz/RSH syndromeB U Fitzky, F F Moebius, H Asaoka, et al.
Nature Genetics|July 23, 2003
Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determinationEdgar A Otto, Bernhard Schermer, Tomoko Obara, et al.
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Showing results (41-50 of 44) with videos related to

Sort By:
Pageof 5
You have reached the last page of results.This site can display upto 44 results.
American Journal of Human Genetics|September 3, 2002
A gene mutated in nephronophthisis and retinitis pigmentosa encodes a novel protein, nephroretinin, conserved in evolutionEdgar Otto, Julia Hoefele, Rainer Ruf, et al.
Trials|April 9, 2014
Evaluation of a practice team-supported exposure training for patients with panic disorder with or without agoraphobia in primary care - study protocol of a cluster randomised controlled superiority trialJochen Gensichen, Thomas S Hiller, Jörg Breitbart, et al.
The Journal of Clinical Investigation|September 19, 2001
7-Dehydrocholesterol-dependent proteolysis of HMG-CoA reductase suppresses sterol biosynthesis in a mouse model of Smith-Lemli-Opitz/RSH syndromeB U Fitzky, F F Moebius, H Asaoka, et al.
Nature Genetics|July 23, 2003
Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determinationEdgar A Otto, Bernhard Schermer, Tomoko Obara, et al.
Pageof 5