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Experimental Cell Research|August 30, 2001
Differences in patterns of activation of MAP kinases induced by oncogenic ras-p21 and insulin in oocytesM Ranginwale, S Smith, J Flom, et al.Cancer Chemotherapy and Pharmacology|August 8, 2001
Plasmid expression of a peptide that selectively blocks oncogenic ras-p21-induced oocyte maturationC Kovac, L Clie, J Morin, et al.Annals of Clinical and Laboratory Science|March 18, 2000
Glutathione-S-Transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinaseA Villafania, K Anwar, S Amar, et al.Breast Cancer Research and Treatment|October 17, 1998
ErbB-2 protein in sera and tumors of breast cancer patientsB Breuer, S Smith, A Thor, et al.Proceedings of the National Academy of Sciences of the United States of America|October 19, 2001
Peptides from the amino terminal mdm-2-binding domain of p53, designed from conformational analysis, are selectively cytotoxic to transformed cellsM Kanovsky, A Raffo, L Drew, et al.Proceedings of the National Academy of Sciences of the United States of America|March 4, 1997
Conformation-dependent phosphorylation of p53V Adler, M R Pincus, T Minamoto, et al.Cancer Chemotherapy and Pharmacology|June 15, 2000
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathwayL Chie, S Amar, H F Kung, et al.Pageof 9