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S J Collins

Showing results (81-90 of 99) with videos related to

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Neurology|March 14, 2007
Novel prion protein gene mutation presenting with subacute PSP-like syndromeD B Rowe, V Lewis, M Needham, et al.
Cancer Research|November 1, 1996
Loss of retinoic acid receptors in mouse skin and skin tumors is associated with activation of the ras(Ha) oncogene and high risk for premalignant progressionN Darwiche, G Scita, C Jones, et al.
Molecular and Cellular Biology|June 1, 1996
An exceptionally conserved transcriptional repressor, CTCF, employs different combinations of zinc fingers to bind diverged promoter sequences of avian and mammalian c-myc oncogenesG N Filippova, S Fagerlie, E M Klenova, et al.
Genes, Chromosomes & Cancer|May 20, 1998
A widely expressed transcription factor with multiple DNA sequence specificity, CTCF, is localized at chromosome segment 16q22.1 within one of the smallest regions of overlap for common deletions in breast and prostate cancersG N Filippova, A Lindblom, L J Meincke, et al.
Biochemistry|March 17, 1999
Familial prion disease mutation alters the secondary structure of recombinant mouse prion protein: implications for the mechanism of prion formationR Cappai, L Stewart, M F Jobling, et al.
American Journal of Hematology|January 1, 1987
Bone marrow transplantation in a patient with myelodysplasia associated with diffuse eosinophilic fasciitisM S Tallman, R W McGuffin, C S Higano, et al.
The American Journal of Pathology|November 7, 1999
Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicityA R White, S J Collins, F Maher, et al.
Journal of Neuroscience Research|September 11, 2001
Involvement of the 5-lipoxygenase pathway in the neurotoxicity of the prion peptide PrP106-126L R Stewart, A R White, M F Jobling, et al.
Neurobiology of Disease|April 13, 2001
Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neuronsA R White, R Guirguis, M W Brazier, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|April 1, 2009
11C-PiB PET studies in typical sporadic Creutzfeldt-Jakob diseaseV L Villemagne, C A McLean, K Reardon, et al.
Pageof 10

Showing results (81-90 of 99) with videos related to

Sort By:
Pageof 10
Neurology|March 14, 2007
Novel prion protein gene mutation presenting with subacute PSP-like syndromeD B Rowe, V Lewis, M Needham, et al.
Cancer Research|November 1, 1996
Loss of retinoic acid receptors in mouse skin and skin tumors is associated with activation of the ras(Ha) oncogene and high risk for premalignant progressionN Darwiche, G Scita, C Jones, et al.
Molecular and Cellular Biology|June 1, 1996
An exceptionally conserved transcriptional repressor, CTCF, employs different combinations of zinc fingers to bind diverged promoter sequences of avian and mammalian c-myc oncogenesG N Filippova, S Fagerlie, E M Klenova, et al.
Genes, Chromosomes & Cancer|May 20, 1998
A widely expressed transcription factor with multiple DNA sequence specificity, CTCF, is localized at chromosome segment 16q22.1 within one of the smallest regions of overlap for common deletions in breast and prostate cancersG N Filippova, A Lindblom, L J Meincke, et al.
Biochemistry|March 17, 1999
Familial prion disease mutation alters the secondary structure of recombinant mouse prion protein: implications for the mechanism of prion formationR Cappai, L Stewart, M F Jobling, et al.
American Journal of Hematology|January 1, 1987
Bone marrow transplantation in a patient with myelodysplasia associated with diffuse eosinophilic fasciitisM S Tallman, R W McGuffin, C S Higano, et al.
The American Journal of Pathology|November 7, 1999
Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicityA R White, S J Collins, F Maher, et al.
Journal of Neuroscience Research|September 11, 2001
Involvement of the 5-lipoxygenase pathway in the neurotoxicity of the prion peptide PrP106-126L R Stewart, A R White, M F Jobling, et al.
Neurobiology of Disease|April 13, 2001
Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neuronsA R White, R Guirguis, M W Brazier, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|April 1, 2009
11C-PiB PET studies in typical sporadic Creutzfeldt-Jakob diseaseV L Villemagne, C A McLean, K Reardon, et al.
Pageof 10