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Proceedings of the National Academy of Sciences of the United States of America|May 10, 1994
Participation of target Fas protein in apoptosis pathway induced by CD4+ Th1 and CD8+ cytotoxic T cellsS T Ju, H Cui, D J Panka, et al.Cellular Immunology|August 1, 1997
CD4+ T cells reactivated with superantigen are both more sensitive to FasL-mediated killing and express a higher level of FasLJ K Wang, B Zhu, S T Ju, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 1, 1995
Fas ligand-mediated cytotoxicity is directly responsible for apoptosis of normal CD4+ T cells responding to a bacterial superantigenR Ettinger, D J Panka, J K Wang, et al.European Journal of Immunology|September 1, 1990
Resistance to lymphoid engraftment in lpr recipients of normal bone marrow: characterization of chimeric stem cell, monocyte and peripheral lymphoid lineagesR M Glaser, A Marshak-RothsteinCurrent Opinion in Immunology|December 1, 1996
B cells in systemic autoimmune disease: recent insights from Fas-deficient mice and menK B Elkon, A Marshak-RothsteinAnnals of the New York Academy of Sciences|April 5, 1997
Unique site of autoantibody production in Fas-deficient miceB A Jacobson, L C Foote, D J Panka, et al.Cellular Immunology|February 1, 1996
Characterization of lpr-derived T cell hybridomas: Fas-deficient hybridomas are deathless, growth-arrested, and cytotoxic upon activationH Cui, M el-Khatib, D H Sherr, et al.Cellular Immunology|February 1, 1996
Regulation of T-cell death genes: selective inhibition of FasL- but not Fas-mediated functionH Cui, D H Sherr, M el-Khatib, et al.European Journal of Immunology|February 1, 1996
Fas (CD95)/Fas ligand interactions regulate antigen-specific, major histocompatibility complex-restricted T/B cell proliferative responsesM Ozdemirli, M El-Khatib, L C Foote, et al.The Journal of Experimental Medicine|April 5, 2000
Opposing effects of transmembrane and soluble Fas ligand expression on inflammation and tumor cell survivalA M Hohlbaum, S Moe, A Marshak-RothsteinPageof 57