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Laboratory Investigation; a Journal of Technical Methods and Pathology|October 26, 1999
Differential expression of individual complement regulators in the brain and choroid plexusS K Singhrao, J W Neal, N K Rushmere, et al.The American Journal of Pathology|September 12, 2000
Spontaneous classical pathway activation and deficiency of membrane regulators render human neurons susceptible to complement lysisS K Singhrao, J W Neal, N K Rushmere, et al.The American Journal of Pathology|January 1, 1997
Expression of the receptor for complement C5a (CD88) is up-regulated on reactive astrocytes, microglia, and endothelial cells in the inflamed human central nervous systemP Gasque, S K Singhrao, J W Neal, et al.Journal of Neuropathology and Experimental Neurology|May 1, 1996
Role of complement in the aetiology of Pick's disease?S K Singhrao, J W Neal, P Gasque, et al.Journal of Immunology (Baltimore, Md. : 1950)|April 8, 1998
The receptor for complement anaphylatoxin C3a is expressed by myeloid cells and nonmyeloid cells in inflamed human central nervous system: analysis in multiple sclerosis and bacterial meningitisP Gasque, S K Singhrao, J W Neal, et al.Experimental and Clinical Immunogenetics|January 1, 1997
Role of complement in inflammation and injury in the nervous systemB P Morgan, P Gasque, S K Singhrao, et al.Neurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration|September 1, 1995
A functional role for corpora amylacea based on evidence from complement studiesS K Singhrao, B P Morgan, J W Neal, et al.Immunology|April 1, 1997
Expression of rat CD59: functional analysis confirms lack of species selectivity and reveals that glycosylation is not required for functionN K Rushmere, S Tomlinson, B P MorganThe Journal of Experimental Medicine|March 28, 1998
Identification of an astrocyte cell population from human brain that expresses perforin, a cytotoxic protein implicated in immune defenseP Gasque, J Jones, S K Singhrao, et al.The Journal of Infection|August 23, 2016
The role of primary infection of Schwann cells in the aetiology of infective inflammatory neuropathiesJ W Neal, P GasquePageof 32