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Brain : a Journal of Neurology|August 16, 2021
Molecular pathology and synaptic loss in primary tauopathies: an 18F-AV-1451 and 11C-UCB-J PET studyNegin Holland, Maura Malpetti, Timothy Rittman, et al.
Neurobiology of Aging|February 25, 2021
In vivo coupling of dendritic complexity with presynaptic density in primary tauopathiesElijah Mak, Negin Holland, P Simon Jones, et al.
Brain : a Journal of Neurology|March 18, 2020
Neuroinflammation and protein aggregation co-localize across the frontotemporal dementia spectrumW Richard Bevan-Jones, Thomas E Cope, P Simon Jones, et al.
Nature Communications|August 31, 2018
Publisher Correction: Variations in Dysbindin-1 are associated with cognitive response to antipsychotic drug treatmentDiego Scheggia, Rosa Mastrogiacomo, Maddalena Mereu, et al.
Acta Neuropathologica Communications|June 13, 2026
α-Synuclein overexpression increases the tropism of pre-formed fibrils and MSA-patient derived seeds towards oligodendrogliaAnne-Line Strange Laursen, Frida Lind-Holm Mogensen, Jonas Folke, et al.
Nature Communications|June 13, 2018
Variations in Dysbindin-1 are associated with cognitive response to antipsychotic drug treatmentDiego Scheggia, Rosa Mastrogiacomo, Maddalena Mereu, et al.
Journal of Nuclear Medicine : Official Publication, Society of Nuclear Medicine|November 19, 2021
In Vivo <sup>18</sup>F-Flortaucipir PET Does Not Accurately Support the Staging of Progressive Supranuclear PalsyMaura Malpetti, Sanne S Kaalund, Kamen A Tsvetanov, et al.
Science (New York, N.Y.)|March 30, 2023
Cytosolic antibody receptor TRIM21 is required for effective tau immunotherapy in mouse modelsAamir S Mukadam, Lauren V C Miller, Annabel E Smith, et al.
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