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Thorax|December 25, 2003
Pulmonary function abnormalities in children with sickle cell diseaseK P Sylvester, R A Patey, P Milligan, et al.Journal of Medical Genetics|February 1, 1986
Meiotic recombination between two polymorphic restriction sites within the beta globin gene clusterJ M Old, C Heath, A Fitches, et al.Journal of Community Genetics|January 12, 2022
The haematology of Jamaicans: red cell indices in HbAA, HbAS, HbAC, and HbA-HPFH genotypesG R Serjeant, B E Serjeant, K P Mason, et al.British Journal of Haematology|November 1, 1993
A base substitution (T-->C) in codon 29 of the alpha 2-globin gene causes alpha thalassaemiaG W Hall, S L Thein, A C Newland, et al.Molecular Biology & Medicine|September 1, 1983
Multiple origins of the sickle mutation: evidence from beta S globin gene cluster polymorphismsJ S Wainscoat, J I Bell, S L Thein, et al.British Journal of Haematology|June 1, 1985
A genetic marker for elevated levels of haemoglobin F in homozygous sickle cell disease?J S Wainscoat, S L Thein, D R Higgs, et al.British Journal of Haematology|July 1, 1991
Novel point mutations leading to type 1 antithrombin deficiency and thrombosisR J Olds, D A Lane, H Ireland, et al.Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|October 31, 1988
Genetic factors as determinants of infectious disease transmission in human communitiesD J Weatherall, J I Bell, J B Clegg, et al.British Journal of Cancer|April 1, 1987
Detection of somatic changes in human cancer DNA by DNA fingerprint analysisS L Thein, A J Jeffreys, H C Gooi, et al.British Journal of Haematology|March 1, 1995
Antithrombins Southport (Leu 99 to Val) and Vienna (Gln 118 to Pro): two novel antithrombin variants with abnormal heparin bindingV Chowdhury, B Mille, R J Olds, et al.Pageof 15