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Virus Research|July 11, 1998
The persistent elevated cytokine mRNA levels in trigeminal ganglia of mice latently infected with HSV-1 are not due to the presence of latency associated transcript (LAT) RNAsD J Carr, W P Halford, L A Veress, et al.
Virology|January 15, 1999
Expression of the first 811 nucleotides of the herpes simplex virus type 1 latency-associated transcript (LAT) partially restores wild-type spontaneous reactivation to a LAT-null mutantB S Drolet, G C Perng, R J Villosis, et al.
Virology|March 17, 1998
The region of the herpes simplex virus type 1 LAT gene involved in spontaneous reactivation does not encode a functional proteinB S Drolet, G C Perng, J Cohen, et al.
Virology|June 15, 1999
Specific and nonspecific immune stimulation of MHC-II-deficient mice results in chronic HSV-1 infection of the trigeminal ganglia following ocular challengeH Ghiasi, G C Perng, F M Hofman, et al.
Investigative Ophthalmology & Visual Science|June 1, 1994
Similarities in regulation of the HSV-1 LAT promoter in corneal and neuronal cellsG C Perng, J C Zwaagstra, H Ghiasi, et al.
Journal of Virology|December 1, 1987
Detection of latency-related viral RNAs in trigeminal ganglia of rabbits latently infected with herpes simplex virus type 1D L Rock, A B Nesburn, H Ghiasi, et al.
Journal of Neurovirology|September 17, 2004
Mapping herpes simplex virus type 1 latency-associated transcript sequences that protect from apoptosis mediated by a plasmid expressing caspase-8W Peng, L Jin, G Henderson, et al.
Virology|October 2, 2012
Sequence and comparative analysis of the genome of HSV-1 strain McKraeG Watson, W Xu, A Reed, et al.
Cancer Gene Therapy|June 8, 2001
A herpes simplex virus type 1 mutant deleted for gamma34.5 and LAT kills glioma cells in vitro and is inhibited for in vivo reactivationK Samoto, G C Perng, M Ehtesham, et al.
Journal of Virology|January 9, 1999
A herpes simplex virus type 1 latency-associated transcript mutant with increased virulence and reduced spontaneous reactivationG C Perng, S M Slanina, A Yukht, et al.
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