Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

S M Gentleman

Showing results (51-60 of 66) with videos related to

Pageof 7
Sort By:
Eye and Brain|May 26, 2017
Differential effects of amyloid-β peptide aggregation status on <i>in vivo</i> retinal neurotoxicityH R Watts, Pjb Anderson, D Ma, et al.
Neuropathology and Applied Neurobiology|November 17, 1999
Overexpression of s100beta in Down's syndrome: correlation with patient age and with beta-amyloid depositionM C Royston, J E McKenzie, S M Gentleman, et al.
Neuropathology and Applied Neurobiology|December 13, 2012
The neuroinflammatory response in humans after traumatic brain injuryC Smith, S M Gentleman, P D Leclercq, et al.
Neuropathology and Applied Neurobiology|June 7, 2000
beta-amyloid (Abeta)42(43), abeta42, abeta40 and apoE immunostaining of plaques in fatal head injuryK Horsburgh, G M Cole, F Yang, et al.
Neuroreport|April 14, 1997
A beta 42 is the predominant form of amyloid beta-protein in the brains of short-term survivors of head injuryS M Gentleman, B D Greenberg, M J Savage, et al.
Neuropathology and Applied Neurobiology|August 28, 2019
Cholinergic deficits and galaninergic hyperinnervation of the nucleus basalis of Meynert in Alzheimer's disease and Lewy body disordersA S Alexandris, L Walker, A K L Liu, et al.
European Archives of Psychiatry and Clinical Neuroscience|December 12, 2012
Neuropathological changes in the nucleus basalis in schizophreniaM R Williams, R Marsh, C D Macdonald, et al.
Brain Pathology (Zurich, Switzerland)|February 11, 1998
Glial-neuronal interactions in Alzheimer's disease: the potential role of a 'cytokine cycle' in disease progressionW S Griffin, J G Sheng, M C Royston, et al.
Cellular and Molecular Neurobiology|March 18, 1999
Is there a genetic basis for the deposition of beta-amyloid after fatal head injury?D I Graham, S M Gentleman, J A Nicoll, et al.
Neuroscience Letters|August 3, 1992
Quantification of beta A4 protein deposition in the medial temporal lobe: a comparison of Alzheimer's disease and senile dementia of the Lewy body typeS M Gentleman, B Williams, M C Royston, et al.
Pageof 7

Showing results (51-60 of 66) with videos related to

Sort By:
Pageof 7
Eye and Brain|May 26, 2017
Differential effects of amyloid-β peptide aggregation status on <i>in vivo</i> retinal neurotoxicityH R Watts, Pjb Anderson, D Ma, et al.
Neuropathology and Applied Neurobiology|November 17, 1999
Overexpression of s100beta in Down's syndrome: correlation with patient age and with beta-amyloid depositionM C Royston, J E McKenzie, S M Gentleman, et al.
Neuropathology and Applied Neurobiology|December 13, 2012
The neuroinflammatory response in humans after traumatic brain injuryC Smith, S M Gentleman, P D Leclercq, et al.
Neuropathology and Applied Neurobiology|June 7, 2000
beta-amyloid (Abeta)42(43), abeta42, abeta40 and apoE immunostaining of plaques in fatal head injuryK Horsburgh, G M Cole, F Yang, et al.
Neuroreport|April 14, 1997
A beta 42 is the predominant form of amyloid beta-protein in the brains of short-term survivors of head injuryS M Gentleman, B D Greenberg, M J Savage, et al.
Neuropathology and Applied Neurobiology|August 28, 2019
Cholinergic deficits and galaninergic hyperinnervation of the nucleus basalis of Meynert in Alzheimer's disease and Lewy body disordersA S Alexandris, L Walker, A K L Liu, et al.
European Archives of Psychiatry and Clinical Neuroscience|December 12, 2012
Neuropathological changes in the nucleus basalis in schizophreniaM R Williams, R Marsh, C D Macdonald, et al.
Brain Pathology (Zurich, Switzerland)|February 11, 1998
Glial-neuronal interactions in Alzheimer's disease: the potential role of a 'cytokine cycle' in disease progressionW S Griffin, J G Sheng, M C Royston, et al.
Cellular and Molecular Neurobiology|March 18, 1999
Is there a genetic basis for the deposition of beta-amyloid after fatal head injury?D I Graham, S M Gentleman, J A Nicoll, et al.
Neuroscience Letters|August 3, 1992
Quantification of beta A4 protein deposition in the medial temporal lobe: a comparison of Alzheimer's disease and senile dementia of the Lewy body typeS M Gentleman, B Williams, M C Royston, et al.
Pageof 7