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The Journal of Biological Chemistry|January 17, 1997
A murine interleukin-4 antagonistic mutant protein completely inhibits interleukin-4-induced cell proliferation, differentiation, and signal transductionS M Grunewald, S Kunzmann, B Schnarr, et al.European Cytokine Network|January 1, 1996
Transmembrane and intracellular signalling by interleukin-4: receptor dimerization and beyondA Duschl, W SebaldInternational Archives of Allergy and Immunology|May 8, 2001
The impact of the route and frequency of antigen exposure on the IgE response in allergyA Nelde, M Teufel, C Hahn, et al.Behring Institute Mitteilungen|June 1, 1995
Antagonistic mutant proteins of interleukin-4A Duschl, T Müller, W SebaldInternational Archives of Allergy and Immunology|September 28, 2001
Upon prolonged allergen exposure IL-4 and IL-4Ralpha knockout mice produce specific IgE leading to anaphylaxisS M Grunewald, M Teufel, K Erb, et al.European Cytokine Network|March 1, 1992
A comparison of assays for the response of primary human T-cells upon stimulation with interleukin-2, interleukin-4 and interleukin-7A Duschl, U Jahn, C Bertling, et al.International Immunology|June 1, 1997
IL-4 receptor complexes containing or lacking the gamma C chain are inhibited by an overlapping set of antagonistic IL-4 mutant proteinsB Schnarr, J Ezernieks, W Sebald, et al.FEBS Letters|April 29, 1996
Interleukin-4 upregulates the heat shock protein Hsp90alpha and enhances transcription of a reporter gene coupled to a single heat shock elementK Metz, J Ezernieks, W Sebald, et al.Journal of Immunology (Baltimore, Md. : 1950)|April 29, 1998
An antagonistic IL-4 mutant prevents type I allergy in the mouse: inhibition of the IL-4/IL-13 receptor system completely abrogates humoral immune response to allergen and development of allergic symptoms in vivoS M Grunewald, A Werthmann, B Schnarr, et al.European Journal of Immunology|July 1, 1997
Mouse macrophage development in the absence of the common gamma chain: defining receptor complexes responsible for IL-4 and IL-13 signalingA Andersson, S M Grunewald, A Duschl, et al.Pageof 12