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Molecular Medicine (Cambridge, Mass.)|May 1, 1995
Normal and expanded Huntington's disease gene alleles produce distinguishable proteins due to translation across the CAG repeatF Persichetti, C M Ambrose, P Ge, et al.Journal of Medical Genetics|July 31, 2007
Factors associated with HD CAG repeat instability in Huntington diseaseV C Wheeler, F Persichetti, S M McNeil, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 1, 1997
Heterogeneous topographic and cellular distribution of huntingtin expression in the normal human neostriatumR J Ferrante, C A Gutekunst, F Persichetti, et al.Brain Research|October 3, 1994
IT15 gene expression in fetal human brainL S Dure, G B Landwehrmeyer, J Golden, et al.Neurobiology of Disease|June 1, 1996
Differential expression of normal and mutant Huntington's disease gene allelesF Persichetti, L Carlee, P W Faber, et al.Annals of Neurology|February 1, 1995
Huntington's disease gene: regional and cellular expression in brain of normal and affected individualsG B Landwehrmeyer, S M McNeil, L S Dure, et al.Human Molecular Genetics|May 1, 1997
Reduced penetrance of the Huntington's disease mutationS M McNeil, A Novelletto, J Srinidhi, et al.Science (New York, N.Y.)|July 21, 1995
Inactivation of the mouse Huntington's disease gene homolog HdhM P Duyao, A B Auerbach, A Ryan, et al.Pageof 1