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S M Snyder

Showing results (11-20 of 18) with videos related to

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Journal of Applied Physiology (Bethesda, Md. : 1985)|May 18, 2001
Renal hypertension prevents run training modification of cardiomyocyte diastolic Ca2+ regulation in male ratsB M Palmer, J M Lynch, S M Snyder, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|December 8, 1998
Shortening and [Ca2+] dynamics of left ventricular myocytes isolated from exercise-trained ratsB M Palmer, A M Thayer, S M Snyder, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|February 4, 1999
Effects of chronic run training on Na+-dependent Ca2+ efflux from rat left ventricular myocytesB M Palmer, J M Lynch, S M Snyder, et al.
The Journal of Biological Chemistry|November 10, 1976
Binding of thyroid hormones and their analogues to thyroxine-binding globulin in human serumS M Snyder, R R Cavalieri, I D Goldfine, et al.
The Journal of Clinical Investigation|March 1, 1990
Familial hypobetalipoproteinemia caused by a mutation in the apolipoprotein B gene that results in a truncated species of apolipoprotein B (B-31). A unique mutation that helps to define the portion of the apolipoprotein B molecule required for the formation of buoyant, triglyceride-rich lipoproteinsS G Young, S T Hubl, R S Smith, et al.
The American Journal of Physiology|December 22, 1999
Chronic run training suppresses alpha-adrenergic response of rat cardiomyocytes and isovolumic left ventricleB M Palmer, M C Olsson, J M Lynch, et al.
The New England Journal of Medicine|June 15, 1989
Familial hypobetalipoproteinemia associated with a mutant species of apolipoprotein B (B-46)S G Young, S T Hubl, D A Chappell, et al.
Journal of Lipid Research|March 1, 1993
Four new mutations in the apolipoprotein B gene causing hypobetalipoproteinemia, including two different frameshift mutations that yield truncated apolipoprotein B proteins of identical lengthS G Young, C R Pullinger, B R Zysow, et al.
Pageof 2

Showing results (11-20 of 18) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 18 results.
Journal of Applied Physiology (Bethesda, Md. : 1985)|May 18, 2001
Renal hypertension prevents run training modification of cardiomyocyte diastolic Ca2+ regulation in male ratsB M Palmer, J M Lynch, S M Snyder, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|December 8, 1998
Shortening and [Ca2+] dynamics of left ventricular myocytes isolated from exercise-trained ratsB M Palmer, A M Thayer, S M Snyder, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|February 4, 1999
Effects of chronic run training on Na+-dependent Ca2+ efflux from rat left ventricular myocytesB M Palmer, J M Lynch, S M Snyder, et al.
The Journal of Biological Chemistry|November 10, 1976
Binding of thyroid hormones and their analogues to thyroxine-binding globulin in human serumS M Snyder, R R Cavalieri, I D Goldfine, et al.
The Journal of Clinical Investigation|March 1, 1990
Familial hypobetalipoproteinemia caused by a mutation in the apolipoprotein B gene that results in a truncated species of apolipoprotein B (B-31). A unique mutation that helps to define the portion of the apolipoprotein B molecule required for the formation of buoyant, triglyceride-rich lipoproteinsS G Young, S T Hubl, R S Smith, et al.
The American Journal of Physiology|December 22, 1999
Chronic run training suppresses alpha-adrenergic response of rat cardiomyocytes and isovolumic left ventricleB M Palmer, M C Olsson, J M Lynch, et al.
The New England Journal of Medicine|June 15, 1989
Familial hypobetalipoproteinemia associated with a mutant species of apolipoprotein B (B-46)S G Young, S T Hubl, D A Chappell, et al.
Journal of Lipid Research|March 1, 1993
Four new mutations in the apolipoprotein B gene causing hypobetalipoproteinemia, including two different frameshift mutations that yield truncated apolipoprotein B proteins of identical lengthS G Young, C R Pullinger, B R Zysow, et al.
Pageof 2