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S Meri

Showing results (141-150 of 157) with videos related to

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Infection and Immunity|May 12, 2001
Complement evasion by Borrelia burgdorferi: serum-resistant strains promote C3b inactivationA Alitalo, T Meri, L Rämö, et al.
The British Journal of Dermatology|May 4, 2019
Tumour-cell-derived complement components C1r and C1s promote growth of cutaneous squamous cell carcinomaP Riihilä, K Viiklepp, L Nissinen, et al.
Annals of Neurology|October 1, 1996
Complement activation in the central nervous system following blood-brain barrier damage in manP J Lindsberg, J Ohman, T Lehto, et al.
European Journal of Clinical Microbiology & Infectious Diseases : Official Publication of the European Society of Clinical Microbiology|September 9, 2011
High pentraxin-3 plasma levels associate with thrombocytopenia in acute Puumala hantavirus-induced nephropathia epidemicaT K Outinen, S Mäkelä, H Huhtala, et al.
Scandinavian Journal of Immunology|April 30, 2018
Distribution of exogenous complement factor H in mice in vivoA R Koskinen, Z-Z Cheng, M C Pickering, et al.
Clinical and Experimental Immunology|June 5, 2003
Release of endogenous anti-inflammatory complement regulators FHL-1 and factor H protects synovial fibroblasts during rheumatoid arthritisM A Friese, T Manuelian, S Junnikkala, et al.
International Immunopharmacology|May 23, 2001
Complement C3b interactions studied with surface plasmon resonance techniqueT S Jokiranta, J Westin, U R Nilsson, et al.
Clinical and Experimental Immunology|April 26, 2006
The C-terminus of complement regulator Factor H mediates target recognition: evidence for a compact conformation of the native proteinM Oppermann, T Manuelian, M Józsi, et al.
Clinical and Experimental Immunology|August 10, 2000
Different regulation of factor H and FHL-1/reconectin by inflammatory mediators and expression of the two proteins in rheumatoid arthritis (RA)M A Friese, J Hellwage, T S Jokiranta, et al.
Plos Pathogens|May 3, 2013
Microbes bind complement inhibitor factor H via a common siteT Meri, H Amdahl, M J Lehtinen, et al.
Pageof 16

Showing results (141-150 of 157) with videos related to

Sort By:
Pageof 16
Infection and Immunity|May 12, 2001
Complement evasion by Borrelia burgdorferi: serum-resistant strains promote C3b inactivationA Alitalo, T Meri, L Rämö, et al.
The British Journal of Dermatology|May 4, 2019
Tumour-cell-derived complement components C1r and C1s promote growth of cutaneous squamous cell carcinomaP Riihilä, K Viiklepp, L Nissinen, et al.
Annals of Neurology|October 1, 1996
Complement activation in the central nervous system following blood-brain barrier damage in manP J Lindsberg, J Ohman, T Lehto, et al.
European Journal of Clinical Microbiology & Infectious Diseases : Official Publication of the European Society of Clinical Microbiology|September 9, 2011
High pentraxin-3 plasma levels associate with thrombocytopenia in acute Puumala hantavirus-induced nephropathia epidemicaT K Outinen, S Mäkelä, H Huhtala, et al.
Scandinavian Journal of Immunology|April 30, 2018
Distribution of exogenous complement factor H in mice in vivoA R Koskinen, Z-Z Cheng, M C Pickering, et al.
Clinical and Experimental Immunology|June 5, 2003
Release of endogenous anti-inflammatory complement regulators FHL-1 and factor H protects synovial fibroblasts during rheumatoid arthritisM A Friese, T Manuelian, S Junnikkala, et al.
International Immunopharmacology|May 23, 2001
Complement C3b interactions studied with surface plasmon resonance techniqueT S Jokiranta, J Westin, U R Nilsson, et al.
Clinical and Experimental Immunology|April 26, 2006
The C-terminus of complement regulator Factor H mediates target recognition: evidence for a compact conformation of the native proteinM Oppermann, T Manuelian, M Józsi, et al.
Clinical and Experimental Immunology|August 10, 2000
Different regulation of factor H and FHL-1/reconectin by inflammatory mediators and expression of the two proteins in rheumatoid arthritis (RA)M A Friese, J Hellwage, T S Jokiranta, et al.
Plos Pathogens|May 3, 2013
Microbes bind complement inhibitor factor H via a common siteT Meri, H Amdahl, M J Lehtinen, et al.
Pageof 16