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Nucleic Acids Research|January 11, 1995
Allele-specific methylation and expression of an imprinted U2af1-rs1 (SP2) geneI Hatada, K Kitagawa, T Yamaoka, et al.Advances in Experimental Medicine and Biology|January 1, 1979
Assay methods for prekallikrein and kininogens and their applicationsY Uchida, S Ohishi, K Tanaka, et al.Pflugers Archiv : European Journal of Physiology|June 13, 1998
The effect of exhaustive exercise on the antioxidant enzyme system in skeletal muscle from calcium-deficient ratsS Ohishi, T Kizaki, T Ookawara, et al.Developmental Cell|November 13, 2001
Diffusion of nodal signaling activity in the absence of the feedback inhibitor Lefty2C Meno, J Takeuchi, R Sakuma, et al.Journal of Medical Genetics|July 29, 1999
Analysis of germline CDKN1C (p57KIP2) mutations in familial and sporadic Beckwith-Wiedemann syndrome (BWS) provides a novel genotype-phenotype correlationW W Lam, I Hatada, S Ohishi, et al.Nature Genetics|October 1, 1996
An imprinted gene p57KIP2 is mutated in Beckwith-Wiedemann syndromeI Hatada, H Ohashi, Y Fukushima, et al.Molecular Cell|October 13, 1999
Mouse Lefty2 and zebrafish antivin are feedback inhibitors of nodal signaling during vertebrate gastrulationC Meno, K Gritsman, S Ohishi, et al.Molecular Cell|March 11, 2000
Left-right asymmetric expression of lefty2 and nodal is induced by a signaling pathway that includes the transcription factor FAST2Y Saijoh, H Adachi, R Sakuma, et al.Human Genetics|October 28, 1997
New p57KIP2 mutations in Beckwith-Wiedemann syndromeI Hatada, A Nabetani, H Morisaki, et al.Human Molecular Genetics|June 1, 1996
Genomic imprinting of human p57KIP2 and its reduced expression in Wilms' tumorsI Hatada, J Inazawa, T Abe, et al.Pageof 5