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S R Wayne Chen

Showing results (61-70 of 142) with videos related to

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Circulation. Arrhythmia and Electrophysiology|June 23, 2012
In situ confocal imaging in intact heart reveals stress-induced Ca(2+) release variability in a murine catecholaminergic polymorphic ventricular tachycardia model of type 2 ryanodine receptor(R4496C+/-) mutationBiyi Chen, Ang Guo, Zhan Gao, et al.
The Journal of Biological Chemistry|October 15, 2003
Residue Gln4863 within a predicted transmembrane sequence of the Ca2+ release channel (ryanodine receptor) is critical for ryanodine interactionRuiwu Wang, Lin Zhang, Jeff Bolstad, et al.
The Journal of Biological Chemistry|May 29, 2008
Reduced threshold for luminal Ca2+ activation of RyR1 underlies a causal mechanism of porcine malignant hyperthermiaDawei Jiang, Wenqian Chen, Jianmin Xiao, et al.
Journal of Molecular and Cellular Cardiology|October 12, 2024
Alda-1 attenuation of binge alcohol-caused atrial arrhythmias through a novel mechanism of suppressed c-Jun N-terminal Kinase-2 activityJiajie Yan, Saugat Khanal, Yuanyuan Cao, et al.
Structure (London, England : 1993)|October 22, 2013
Conformational dynamics inside amino-terminal disease hotspot of ryanodine receptorXiaowei Zhong, Ying Liu, Li Zhu, et al.
The FEBS Journal|June 24, 2019
Diminished inhibition and facilitated activation of RyR2-mediated Ca<sup>2+</sup> release is a common defect of arrhythmogenic calmodulin mutationsMads T Søndergaard, Yingjie Liu, Malene Brohus, et al.
Molecular Pharmacology|June 16, 2005
The Gln4863Ala mutation within a putative, pore-lining trans-membrane helix of the cardiac ryanodine receptor channel alters both the kinetics of ryanoid interaction and the subsequent fractional conductanceKishani M Ranatunga, Tracy M Moreno-King, Bhavna Tanna, et al.
The Biochemical Journal|June 11, 2013
The CPVT-associated RyR2 mutation G230C enhances store overload-induced Ca2+ release and destabilizes the N-terminal domainsYingjie Liu, Lynn Kimlicka, Florian Hiess, et al.
Journal of Medicinal Chemistry|October 16, 2013
Novel carvedilol analogues that suppress store-overload-induced Ca2+ releaseChris D Smith, Aixia Wang, Kannan Vembaiyan, et al.
Europace : European Pacing, Arrhythmias, and Cardiac Electrophysiology : Journal of the Working Groups on Cardiac Pacing, Arrhythmias, and Cardiac Cellular Electrophysiology of the European Society of Cardiology|June 30, 2023
RYR2-ryanodinopathies: from calcium overload to calcium deficiencyChristian Steinberg, Thomas M Roston, Christian van der Werf, et al.
Pageof 15

Showing results (61-70 of 142) with videos related to

Sort By:
Pageof 15
Circulation. Arrhythmia and Electrophysiology|June 23, 2012
In situ confocal imaging in intact heart reveals stress-induced Ca(2+) release variability in a murine catecholaminergic polymorphic ventricular tachycardia model of type 2 ryanodine receptor(R4496C+/-) mutationBiyi Chen, Ang Guo, Zhan Gao, et al.
The Journal of Biological Chemistry|October 15, 2003
Residue Gln4863 within a predicted transmembrane sequence of the Ca2+ release channel (ryanodine receptor) is critical for ryanodine interactionRuiwu Wang, Lin Zhang, Jeff Bolstad, et al.
The Journal of Biological Chemistry|May 29, 2008
Reduced threshold for luminal Ca2+ activation of RyR1 underlies a causal mechanism of porcine malignant hyperthermiaDawei Jiang, Wenqian Chen, Jianmin Xiao, et al.
Journal of Molecular and Cellular Cardiology|October 12, 2024
Alda-1 attenuation of binge alcohol-caused atrial arrhythmias through a novel mechanism of suppressed c-Jun N-terminal Kinase-2 activityJiajie Yan, Saugat Khanal, Yuanyuan Cao, et al.
Structure (London, England : 1993)|October 22, 2013
Conformational dynamics inside amino-terminal disease hotspot of ryanodine receptorXiaowei Zhong, Ying Liu, Li Zhu, et al.
The FEBS Journal|June 24, 2019
Diminished inhibition and facilitated activation of RyR2-mediated Ca<sup>2+</sup> release is a common defect of arrhythmogenic calmodulin mutationsMads T Søndergaard, Yingjie Liu, Malene Brohus, et al.
Molecular Pharmacology|June 16, 2005
The Gln4863Ala mutation within a putative, pore-lining trans-membrane helix of the cardiac ryanodine receptor channel alters both the kinetics of ryanoid interaction and the subsequent fractional conductanceKishani M Ranatunga, Tracy M Moreno-King, Bhavna Tanna, et al.
The Biochemical Journal|June 11, 2013
The CPVT-associated RyR2 mutation G230C enhances store overload-induced Ca2+ release and destabilizes the N-terminal domainsYingjie Liu, Lynn Kimlicka, Florian Hiess, et al.
Journal of Medicinal Chemistry|October 16, 2013
Novel carvedilol analogues that suppress store-overload-induced Ca2+ releaseChris D Smith, Aixia Wang, Kannan Vembaiyan, et al.
Europace : European Pacing, Arrhythmias, and Cardiac Electrophysiology : Journal of the Working Groups on Cardiac Pacing, Arrhythmias, and Cardiac Cellular Electrophysiology of the European Society of Cardiology|June 30, 2023
RYR2-ryanodinopathies: from calcium overload to calcium deficiencyChristian Steinberg, Thomas M Roston, Christian van der Werf, et al.
Pageof 15