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S Redwood

Showing results (11-20 of 85) with videos related to

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The Journal of Biological Chemistry|May 12, 2000
Altered regulatory properties of human cardiac troponin I mutants that cause hypertrophic cardiomyopathyK Elliott, H Watkins, C S Redwood
Academic Medicine : Journal of the Association of American Medical Colleges|June 27, 1998
Improving fourth-year students' complex interviewing skillsN W Van Winkle, M Rosenbaum, S Redwood
Journal of Muscle Research and Cell Motility|October 20, 2006
A revised method of troponin exchange in permeabilised cardiac trabeculae using vanadate: functional consequences of a HCM-causing mutation in troponin ILaura C Preston, Hugh Watkins, Charles S Redwood
Biochemical and Biophysical Research Communications|August 30, 1988
Reversal of caldesmon function by anti-caldesmon antibodies confirms its role in the calcium regulation of vascular smooth muscle thin filamentsS B Marston, C S Redwood, W Lehman
Biofizika|January 14, 2009
[Effect of mutation Arg9lGly on the thermal stability of beta-tropomyosin]I A Nevzorov, C S Redwood, D I Levitskiĭ
Archives of Biochemistry and Biophysics|March 25, 2014
The E117K mutation in β-tropomyosin disturbs concerted conformational changes of actomyosin in muscle fibersOlga E Karpicheva, Charles S Redwood, Yurii S Borovikov
Cardiovascular Research|January 1, 2000
Properties of mutant contractile proteins that cause hypertrophic cardiomyopathyC S Redwood, J C Moolman-Smook, H Watkins
Biochemical and Biophysical Research Communications|June 20, 2007
DCM troponin C mutant Gly159Asp blunts the response to troponin phosphorylationLaura C Preston, Christopher C Ashley, Charles S Redwood
FEBS Letters|July 19, 1993
The functional effects of mutations Thr673-->Asp and Ser702-->Asp at the Pro-directed kinase phosphorylation sites in the C-terminus of chicken gizzard caldesmonC S Redwood, S B Marston, N B Gusev
Circulation Research|October 13, 2007
Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filamentsPaul Robinson, Peter J Griffiths, Hugh Watkins, et al.
Pageof 9

Showing results (11-20 of 85) with videos related to

Sort By:
Pageof 9
The Journal of Biological Chemistry|May 12, 2000
Altered regulatory properties of human cardiac troponin I mutants that cause hypertrophic cardiomyopathyK Elliott, H Watkins, C S Redwood
Academic Medicine : Journal of the Association of American Medical Colleges|June 27, 1998
Improving fourth-year students' complex interviewing skillsN W Van Winkle, M Rosenbaum, S Redwood
Journal of Muscle Research and Cell Motility|October 20, 2006
A revised method of troponin exchange in permeabilised cardiac trabeculae using vanadate: functional consequences of a HCM-causing mutation in troponin ILaura C Preston, Hugh Watkins, Charles S Redwood
Biochemical and Biophysical Research Communications|August 30, 1988
Reversal of caldesmon function by anti-caldesmon antibodies confirms its role in the calcium regulation of vascular smooth muscle thin filamentsS B Marston, C S Redwood, W Lehman
Biofizika|January 14, 2009
[Effect of mutation Arg9lGly on the thermal stability of beta-tropomyosin]I A Nevzorov, C S Redwood, D I Levitskiĭ
Archives of Biochemistry and Biophysics|March 25, 2014
The E117K mutation in β-tropomyosin disturbs concerted conformational changes of actomyosin in muscle fibersOlga E Karpicheva, Charles S Redwood, Yurii S Borovikov
Cardiovascular Research|January 1, 2000
Properties of mutant contractile proteins that cause hypertrophic cardiomyopathyC S Redwood, J C Moolman-Smook, H Watkins
Biochemical and Biophysical Research Communications|June 20, 2007
DCM troponin C mutant Gly159Asp blunts the response to troponin phosphorylationLaura C Preston, Christopher C Ashley, Charles S Redwood
FEBS Letters|July 19, 1993
The functional effects of mutations Thr673-->Asp and Ser702-->Asp at the Pro-directed kinase phosphorylation sites in the C-terminus of chicken gizzard caldesmonC S Redwood, S B Marston, N B Gusev
Circulation Research|October 13, 2007
Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filamentsPaul Robinson, Peter J Griffiths, Hugh Watkins, et al.
Pageof 9