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S Redwood

Showing results (31-40 of 85) with videos related to

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FEBS Letters|September 17, 1990
The functional properties of full length and mutant chicken gizzard smooth muscle caldesmon expressed in Escherichia coliC S Redwood, S B Marston, J Bryan, et al.
Molecular & General Genetics : MGG|August 27, 1996
Human DNA topoisomerases II alpha and II beta can functionally substitute for yeast TOP2 in chromosome segregation and recombinationS Jensen, C S Redwood, J R Jenkins, et al.
Heart (British Cardiac Society)|October 12, 2005
Does a well developed collateral circulation predispose to restenosis after percutaneous coronary intervention? An intravascular ultrasound studyD Perera, P Postema, R Rashid, et al.
Biochemical and Biophysical Research Communications|February 19, 2009
Dilated cardiomyopathy mutations in alpha-tropomyosin inhibit its movement during the ATPase cycleYurii S Borovikov, Olga E Karpicheva, Galina A Chudakova, et al.
Archives of Biochemistry and Biophysics|August 4, 2009
The effect of the dilated cardiomyopathy-causing mutation Glu54Lys of alpha-tropomyosin on actin-myosin interactions during the ATPase cycleYurii S Borovikov, Olga E Karpicheva, Stanislava V Avrova, et al.
Circulation|May 1, 1995
Angiographic stenosis progression and coronary events in patients with 'stabilized' unstable anginaL Chen, M R Chester, S Redwood, et al.
Environmental Science & Technology|May 6, 2005
Characterization of humic substances by environmental scanning electron microscopyPaul S Redwood, Jamie R Lead, Roy M Harrison, et al.
The American Journal of Cardiology|January 15, 1996
Use of ventricular premature complexes for risk stratification after acute myocardial infarction in the thrombolytic eraD J Statters, M Malik, S Redwood, et al.
Biochimica Et Biophysica Acta|December 29, 2015
Myopathy-causing Q147P TPM2 mutation shifts tropomyosin strands further towards the open position and increases the proportion of strong-binding cross-bridges during the ATPase cycleOlga E Karpicheva, Armen O Simonyan, Nadezhda V Kuleva, et al.
International Journal of Molecular Sciences|October 17, 2020
Looking for Targets to Restore the Contractile Function in Congenital Myopathy Caused by Gln<sup>147</sup>Pro TropomyosinOlga E Karpicheva, Armen O Simonyan, Nikita A Rysev, et al.
Pageof 9

Showing results (31-40 of 85) with videos related to

Sort By:
Pageof 9
FEBS Letters|September 17, 1990
The functional properties of full length and mutant chicken gizzard smooth muscle caldesmon expressed in Escherichia coliC S Redwood, S B Marston, J Bryan, et al.
Molecular & General Genetics : MGG|August 27, 1996
Human DNA topoisomerases II alpha and II beta can functionally substitute for yeast TOP2 in chromosome segregation and recombinationS Jensen, C S Redwood, J R Jenkins, et al.
Heart (British Cardiac Society)|October 12, 2005
Does a well developed collateral circulation predispose to restenosis after percutaneous coronary intervention? An intravascular ultrasound studyD Perera, P Postema, R Rashid, et al.
Biochemical and Biophysical Research Communications|February 19, 2009
Dilated cardiomyopathy mutations in alpha-tropomyosin inhibit its movement during the ATPase cycleYurii S Borovikov, Olga E Karpicheva, Galina A Chudakova, et al.
Archives of Biochemistry and Biophysics|August 4, 2009
The effect of the dilated cardiomyopathy-causing mutation Glu54Lys of alpha-tropomyosin on actin-myosin interactions during the ATPase cycleYurii S Borovikov, Olga E Karpicheva, Stanislava V Avrova, et al.
Circulation|May 1, 1995
Angiographic stenosis progression and coronary events in patients with 'stabilized' unstable anginaL Chen, M R Chester, S Redwood, et al.
Environmental Science & Technology|May 6, 2005
Characterization of humic substances by environmental scanning electron microscopyPaul S Redwood, Jamie R Lead, Roy M Harrison, et al.
The American Journal of Cardiology|January 15, 1996
Use of ventricular premature complexes for risk stratification after acute myocardial infarction in the thrombolytic eraD J Statters, M Malik, S Redwood, et al.
Biochimica Et Biophysica Acta|December 29, 2015
Myopathy-causing Q147P TPM2 mutation shifts tropomyosin strands further towards the open position and increases the proportion of strong-binding cross-bridges during the ATPase cycleOlga E Karpicheva, Armen O Simonyan, Nadezhda V Kuleva, et al.
International Journal of Molecular Sciences|October 17, 2020
Looking for Targets to Restore the Contractile Function in Congenital Myopathy Caused by Gln<sup>147</sup>Pro TropomyosinOlga E Karpicheva, Armen O Simonyan, Nikita A Rysev, et al.
Pageof 9